Published online Jun 6, 2021. doi: 10.12998/wjcc.v9.i16.3880
Peer-review started: January 22, 2021
First decision: February 28, 2021
Revised: March 10, 2021
Accepted: March 23, 2021
Article in press: March 23, 2021
Published online: June 6, 2021
Processing time: 112 Days and 1 Hours
Colorectal cancer (CRC) is one of the most malignant gastrointestinal cancers worldwide. The liver is the most important metastatic target organ, and liver metastasis is the leading cause of death in CRC patients.
There is still a lack of diagnostic or prognostic markers for liver metastasis (LM) of CRC. Therefore, it is very important to identify the diagnostic or prognostic markers for LM of CRC to improve the clinical outcomes.
This study aimed to explore the highly differentially expressed genes (HDEGs) and prognostic marker for LM of CRC.
Three NCBI Gene Expression Omnibus (GEO) datasets were utilized to identify a set of HDEGs. These significantly HDEGs of the three GEO datasets take the intersection genes and these intersection genes were screened through an online tool to explore their prognostic value. TIMER and R package were utilized to investigate potential immune functions of HDEGs and gene set enrichment analysis was performed to explore their possible impact on CRC.
APOC1 is one of 47 HDEGs in three GEO datasets for LM of CRC and showed significantly different expression between different N and T stages in the TCGA database. APOC1 mRNA was strongly upregulated in cancer tissues compared with normal tissues, as confirmed by quantitative real-time polymerase chain reaction. The prognostic value of APOC1 for overall survival and disease-free survival in CRC was revealed with PrognoScan and GEPIA2. APOC1 also has a close relationship with immune infiltration showed with TIMER.
APOC1 is a potential biomarker that is associated with both the diagnosis and prognosis of liver metastases of colorectal cancer.
Future work and basic research should be performed to confirm these findings of APOC1 and to verify the related potential regulatory mechanisms in vitro and in vivo.