Published online Dec 16, 2021. doi: 10.12998/wjcc.v9.i35.10948
Peer-review started: April 25, 2021
First decision: June 3, 2021
Revised: June 18, 2021
Accepted: August 30, 2021
Article in press: August 30, 2021
Published online: December 16, 2021
Processing time: 229 Days and 0.9 Hours
Peptic ulcer is a relatively common chronic gastrointestinal disorder most frequently caused by Helicobacter pylori infection or long-term non-steroid anti-inflammatory drug administration. Severe cases are often complicated by bleeding, which can exacerbate symptoms, cause anemia, and lead to life-threatening hemorrhage. Hypersecretion of gastric acid increases the risk and severity of peptic ulcer. Gastric acid secretion is controlled by the hormones gastrin-17 (G-17), pepsinogen I (PGI), and pepsinogen II (PGII), suggesting that these factors may be predictive of bleeding among peptic ulcer patients.
If left untreated, peptic ulcer can result in gastrointestinal bleeding and gastric tumors. Therefore, accessible biomarkers such as serum factors predictive of bleeding risk are important tools for early diagnosis and treatment guidance, and may reduce the incidence of severe complications.
To examine if serum G-17, PGI, PGII, and (or) PGI/PGII ratio (PGR) can predict bleeding risk among peptic ulcer patients.
We compared serum G-17, PGI, PGII, and PGR between 199 peptic ulcer patients with bleeding (n = 92) or without bleeding (n = 107), and then assessed the efficacy of each factor for bleeding prediction by receiver operating characteristic curve analysis.
Serum G-17 was significantly higher among peptic ulcer patients with upper gastrointestinal bleeding, whereas serum PGI, PGII, and PGR did not differ significantly between bleeding and non-bleeding patients. A serum G-17 greater than 9.86 pmol/L distinguished bleeding from non-bleeding patients with 90.2% sensitivity and 68.2% specificity.
Elevated serum G-17 is predictive of bleeding risk among peptic ulcer patients. Lowering serum G-17 should be a major goal of clinical intervention.
Prospective studies are warranted to assess if elevated serum G-17 can predict bleeding complication prior to disease onset. The development of drugs able to regulate G-17 secretion is also desired to help reduce bleeding risk.