Published online Dec 6, 2020. doi: 10.12998/wjcc.v8.i23.5952
Peer-review started: August 28, 2020
First decision: September 29, 2020
Revised: October 3, 2020
Accepted: October 20, 2020
Article in press: October 20, 2020
Published online: December 6, 2020
Processing time: 97 Days and 15.4 Hours
Coronavirus disease 2019 (COVID-19) has spread to many countries and regions all over the world and has become a worldwide public health event. COVID-19 is an acute infectious disease caused by a new coronavirus [severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2)], which is clinically categorized into mild, moderate, severe, and critical illness. Severe and critically ill patients progress rapidly with dyspnea, hypoxemia, and even life-threatening complications such as multiple organ dysfunction syndrome, sepsis, and shock. At present, there is no significant and effective drug for severe and critical patients with COVID-19. Glucocorticoids have been used in the treatment of SARS, Middle East respiratory syndrome, influenza A, and other infectious respiratory diseases worldwide, but their efficacy is still controversial.
In clinical practice, some severe and critical patients with COVID-19 benefit from the application of glucocorticoids, but some patients have various adverse effects. Therefore, whether glucocorticoids should be used in patients with COVID-19 and how to use them are a problem worthy of discussion.
The main objective of this study was to determine the effect of methylprednisolone in severe and critical patients with COVID-19.
One hundred and two severe and critically ill patients with COVID-19 were divided into treatment (69, 67.6%) and control groups (33, 32.4%). In the treatment group, methylprednisolone was intravenously administered at 0.75-1.5 mg/kg/d, usually for less than 14 d. We compared the general information, underlying diseases, laboratory examination indexes, and mortality of the two groups. The log-rank test and the Kaplan–Meier survival curve were used to explore the difference in mortality between the two groups, and the COX regression equation was used to correct the variables with differences.
The treatment group patients had higher aspartic acid aminotransferase (P < 0.01), globulin (P < 0.01), hydroxybutyrate dehydrogenase (P < 0.01), and lactate dehydrogenase (P < 0.01). Twenty-nine (78.4%) of patients in the treatment group died as opposed to 40 (61.5%) in the control group. The mortality was higher than that of the control group. And the results showed that methylprednisolone treatment did not improve prognosis.
Methylprednisolone treatment does not improve prognosis in severe and critical COVID-19 patients.
Methylprednisolone treatment in severe and critically ill patients with COVID-19 should be comprehensively evaluated and used with caution.