Published online Jul 6, 2020. doi: 10.12998/wjcc.v8.i13.2727
Peer-review started: January 16, 2020
First decision: April 14, 2020
Revised: April 26, 2020
Accepted: June 29, 2020
Article in press: June 29, 2020
Published online: July 6, 2020
Processing time: 172 Days and 18.6 Hours
The eosinophilic phenotype accounts for 20%-40% of chronic obstructive pulmonary disease (COPD) exacerbations. The peripheral blood eosinophil count (PBEC) is a well-established predictor of the length of hospital stay, steroid response, prognosis, and readmission rate. Additionally, previous research supported the tendency of non-infectious inflammation in the eosinophilic group.
Some characteristics of patients with eosinophilic COPD exacerbation, such as demographics, comorbidities, lung function, etc., are inconsistent across studies. Studies on the role of eosinophilia primarily focused on the Caucasian race. A few studies investigated eosinophilic COPD exacerbation in Asian populations.
We aimed to study the role of peripheral eosinophilia in hospitalized patients with COPD exacerbation in Taiwan.
From January 2014 to May 2017, patients with COPD exacerbation hospitalized in Taipei Tzu Chi Hospital were retrospectively stratified into two groups according to their peripheral eosinophil count: The EOS group (eosinophil count ≥ 2%) and the non-EOS group (eosinophil count < 2%).
A total of 625 patients were recruited, with 176 patients (28.2%) in the eosinophilic group. The eosinophilic group showed a lower prevalence of infection, lower cumulative doses of prednisolone equivalents, shorter length of hospital stay, and higher number of COPD-related readmissions than the non-EOS group. There were significantly linear correlations between eosinophil percentage and number of readmissions and between eosinophil percentage and length of hospital stay (P < 0.001, Pearson's r = 0.147; P = 0.031, Pearson's r = -0.086, respectively).
The PBEC had a positive linear correlation with the number of readmissions. Eosinophilia is a predictor of steroid response and non-infectious inflammation.
We should strengthen the management of comorbidities and optimization of inhaled medications to reduce the high readmission risk in the EOS group. Routine survey of PBEC for acute COPD exacerbation is warranted to reduce the notorious side effects of steroids. With meticulous exclusion of possible infections, we could avoid empirical antibiotic therapy since the EOS group has a non-infectious nature. Effective target therapy for eosinophilic COPD exacerbation is urgently required.