Published online Jan 26, 2020. doi: 10.12998/wjcc.v8.i2.325
Peer-review started: October 31, 2019
First decision: November 19, 2019
Revised: December 9, 2019
Accepted: December 13, 2019
Article in press: December 13, 2019
Published online: January 26, 2020
Processing time: 78 Days and 1.1 Hours
False tendon is a common intraventricular anatomical variation. It refers to a fibroid or fibromuscular structure that exists in the ventricle besides the normal connection of papillary muscle and mitral or tricuspid valve. A large number of clinical studies have suggested that there is a significant correlation between false tendons and premature ventricular complexes. However, few studies have verified this correlation during radiofrequency catheter ablation of premature ventricular complexes.
A 45-year-old male was admitted to receive radiofrequency ablation for symptomatic premature ventricular complexes. A three-dimensional model of the left ventricle was established by intracardiac echocardiography using the CartoSoundTM mapping system. In addition to the left anterior papillary muscle, the posterior papillary muscle was mapped. False tendons were found at the base of the interventricular septum, and the other end was connected to the left ventricular free wall near the apex. An irrigated touch force catheter was advanced into the left ventricle via the retrograde approach. The earliest activation site was marked at the interventricular septum attachment of the false tendons and was successfully ablated.
This case verified that false tendons can cause premature ventricular complexes and may be cured by radiofrequency ablation guided by intracardiac echocardiography with the CartoSoundTM system.
Core tip: We report a case of successful radiofrequency catheter ablation of premature ventricular complexes associated with left ventricular false tendons. Intracardiac echocardiography with the CartoSoundTM system was used to demonstrate, for the first time, that the occurrence of ventricular premature complexes was associated with false tendons. No classical Purkinje potential and special potential were observed following interventricular septum attachment of false tendons, the local potential of the target region in this patient, and the ectopic excitability resulting in mechanical traction at the false tendon attachment site of the interventricular septum may be a possible mechanism for these premature ventricular complexes.