Published online Sep 26, 2018. doi: 10.12998/wjcc.v6.i10.344
Peer-review started: April 23, 2018
First decision: May 23, 2018
Revised: June 8, 2018
Accepted: June 27, 2018
Article in press: June 28, 2018
Published online: September 26, 2018
Processing time: 156 Days and 1 Hours
Microcystins (MCs) are produced by certain bloom-forming cyanobacteria that can induce toxicity in various organs, including renal toxicity, reproductive toxicity, cardiotoxicity, and immunosuppressive effects. It has been a significant global environmental issue due to its harm to the aquatic environment and human health. Numerous investigators have demonstrated that MC exposure can induce a widespread epidemic of enterogastritis with symptoms similar to food poisoning in areas close to lakes. Both in vivo and in vitro studies have provided evidence of positive associations between MC exposure and gastrointestinal toxicity. The toxicity of MCs on the gastrointestinal tract is multidimensional. MCs can affect gastrointestinal barrier function and shift the structure of gut microbiota in different gut regions. Furthermore, MCs can inhibit the secretion of gastrointestinal digestive enzymes and the release of inflammatory cytokines, which affects the expression of immune-related genes in the intestine. The damage of the intestine is closely correlated to MC exposure because the intestine is the main site for the digestion and absorption of nutrients. The damage to the gastrointestinal tract due to MCs was summarized from different aspects, which can be used as a foundation for further exploration of molecular damage mechanisms.
Core tip: First, the gastrointestinal toxicity of microcystins (MCs) on a population was described. Second, the concentration or localization of MCs in the small intestine after exposure to various concentrations and different time points, as well as after the depuration of MCs in the intestine, was summarized. Third, the change in morphologic pathology and other effects such as oxidative stress, immunotoxicity, digestive enzymes, and gut microbiota in the intestine with exposure to MCs were discussed. Further challenges that need to be addressed were also summarized.