Published online May 6, 2025. doi: 10.12998/wjcc.v13.i13.101438
Revised: December 10, 2024
Accepted: December 23, 2024
Published online: May 6, 2025
Processing time: 119 Days and 9.1 Hours
The relationship between genetics and infectious diseases is important in shaping our understanding of disease susceptibility, progression, and treatment. Recent research shows the impact of genetic variations, such as heme-oxygenase promoter length, on diseases like malaria and sepsis, revealing both protective and inconclusive effects. Studies on vaccine responses highlight genetic markers like human leukocyte antigens, emphasizing the potential for personalized immunization strategies. The ongoing battle against drug-resistant tuberculosis (TB) illustrates the complexity of genomic variants in predicting resistance, highlighting the need for integrated diagnostic tools. Additionally, genome-wide association studies reveal antibiotic resistance mechanisms in bacterial genomes, while host genetic polymorphisms, such as those in solute carrier family 11 member 1 and vitamin D receptor, demonstrate their role in TB susceptibility. Advanced techniques like metagenomic next-generation sequencing promise detailed pathogen detection but face challenges in cost and accessibility. A case report involving a highly virulent Mycobacterium TB strain with the pks1 gene further highlights the need for genetic insights in understanding disease severity and developing targeted interventions. This evolving landscape emphasizes the role of genetics in infectious diseases, while also addressing the need for standardized studies and accessible technologies.
Core Tip: Genetics significantly impacts infectious disease outcomes, from susceptibility to treatment responses. Key genetic variations, such as those in heme-oxygenase-1 and vaccine-related markers, influence how individuals respond to infections and vaccinations. The study of drug-resistant tuberculosis highlights the complex role of genomic variants, requiring advanced diagnostic tools. Genome-wide association studies and host polymorphisms, like solute carrier family 11 member 1 and vitamin D receptor, provide insights into disease mechanisms and resistance.