Published online Apr 16, 2024. doi: 10.12998/wjcc.v12.i11.1881
Peer-review started: February 14, 2024
First decision: March 2, 2024
Revised: March 6, 2024
Accepted: March 22, 2024
Article in press: March 22, 2024
Published online: April 16, 2024
Processing time: 57 Days and 4.6 Hours
Hepatolithiasis (HL) poses a significant risk for cholangiocarcinoma (CCA) development, with reported incidences ranging from 5%-13%. Risk factors include older age, smoking, hepatitis B infection, and prolonged HL duration. Chronic inflammation and mechanical stress on the biliary epithelium contribute to CCA pathogenesis. Hepatectomy reduces CCA risk by removing stones and atrophic liver segments. However, residual stones and incomplete removal increase CCA risk. Kim et al identified carbohydrate antigen 19-9, carcinoembryonic antigen, and stone laterality as CCA risk factors, reaffirming the importance of complete stone removal. Nonetheless, challenges remain in preventing CCA recurrence post-surgery. Longer-term studies are needed to elucidate CCA risk factors further.
Core Tip: Hepatolithiasis (HL) poses a significant risk for cholangiocarcinoma (CCA), with factors like stone location, recurrence, and incomplete removal influencing risk. While hepatectomy reduces CCA risk, residual stones and incomplete removal pose challenges. Kim et al's study identifies carbohydrate antigen 19-9, carcinoembryonic antigen, and stone laterality as CCA risk factors, supporting prior findings. Nonetheless, discrepancies in bile duct stricture's impact on CCA risk highlight the need for further research. Understanding these factors aids in refining CCA risk assessment and optimizing management strategies for HL-associated CCA.