Published online Aug 26, 2022. doi: 10.12998/wjcc.v10.i24.8703
Peer-review started: March 4, 2022
First decision: June 7, 2022
Revised: June 16, 2022
Accepted: July 21, 2022
Article in press: July 21, 2022
Published online: August 26, 2022
Processing time: 164 Days and 17.3 Hours
Tofacitinib is an oral Janus kinase (JAK) inhibitor that is currently approved by the United States Food and Drug Administration for the treatment of rheumatoid arthritis (RA). Varicella zoster virus reactivation leading to herpes zoster (HZ) is an adverse effect of this drug; however, recurrent HZ at the same site is a rare clinical condition.
A 70-year-old female RA patient had undergone 1-year of tofacitinib treatment (10 mg daily). About 1 mo after initiation of oral tofacitinib, she developed blisters on the left flank and abdomen and was diagnosed with HZ; antiviral therapy with acyclovir was resolutory. However, 5 d prior to presentation at our hospital, erythema and blisters with severe pain recurred at the same site. Small clustered blisters and bullous were visible on the left lumbar abdomen and perineum, with a pain score of 8 (visual analogue scale). Antiviral, nutritional supplement, analgesic and other treatments led to healing but over an atypically long period (approximately 26 d, vs approximately 1 wk). HZ is a common and serious adverse reaction of JAK inhibitors, but it rarely recurs. Our patient’s experience of HZ recurrence at the same site, with a wider affected area, more severe pain and longer healing period, is inconsistent with previous reports.
Same-anatomical site HZ recurrence may occur during oral tofacitinib treatment, with more severe clinical manifestations than in the initial occurrence.
Core Tip: Herpes zoster (HZ) is caused by the reactivation of varicella zoster virus. We report a case of recurrent HZ in a patient with rheumatoid arthritis treated with oral tofacitinib. This patient’s HZ recurred at the same site, with a wider affected area and more significant pain than the first occurrence. After antiviral therapy, the rash slowly resolved. The characteristics of this case are different to those of HZ induced by tofacitinib summarized and analyzed in the literature. We hope that our report will prompt clinicians to standardize the diagnosis and treatment of HZ during tofacitinib therapy.