Published online Nov 15, 2021. doi: 10.4251/wjgo.v13.i11.1741
Peer-review started: June 3, 2021
First decision: June 30, 2021
Revised: July 10, 2021
Accepted: August 24, 2021
Article in press: August 24, 2021
Published online: November 15, 2021
Processing time: 162 Days and 3.6 Hours
Spasmolytic polypeptide-expressing metaplasia (SPEM) is the first metaplastic lesion to evolve and probably progresses to intestinal metaplasia.
Our group proved that Yiwei Xiaoyu granules (YWXY) could improve the mucosa atrophy, intestinal metaplasia and dysplasia of chronic gastric gastritis in a clinical trial, while the specific mechanism of YWXY still remains largely unknown.
To elucidate microRNA-7-mediated preventive and inhibitive effects of YWXY in SPEM lesions.
Gastric mucosa biopsies were collected both in human and in a tamoxifen-induced SPEM mouse model. Then immunohistochemistry and immunofluorescence were performed to validate the SPEM lesions, and the potential mechanism was investigated. RNA transcripts were detected with reverse transcription-quantitative polymerase chain reaction.
We showed evidence that microRNA-7 downregulation was an early event in the cascade from metaplasia to gastric cancer and that it contributed to the establishment of an intestinal expression profile through regulation of trefoil factor 2 both in human gastric mucosa and an in vivo model. We validated that YWXY had the ability of inhibiting the cell proliferation and restoring the expression of microRNA-7 by mediating trefoil factor 2 in SPEM lesions.
To the best of our knowledge, it is the first time to use the SPEM mouse model to uncover the effectiveness and potential mechanism of Chinese medicine for the precursor of gastric adenocarcinoma.
Nevertheless, the detailed mechanism of YWXY to prevent and inhibit the development and progression of SPEM lesions should be examined carefully in our next experiment. We believe it shows great potential for drug development to prevent and treat precancerous lesions.