Published online Feb 21, 2015. doi: 10.3748/wjg.v21.i7.2183
Peer-review started: June 1, 2014
First decision: July 21, 2014
Revised: August 12, 2014
Accepted: September 5, 2014
Article in press: September 5, 2014
Published online: February 21, 2015
Processing time: 255 Days and 21.8 Hours
AIM: To perform a profiling analysis of changes in intestinal microRNA (miRNA) expression during hypothermic circulatory arrest (HCA).
METHODS: A total of eight piglets were randomly divided into HCA and sham operation (SO) groups. Under general anesthesia, swine in the HCA group were subjected to hypothermic cardiopulmonary bypass at 24 °C followed by 80 min of circulatory arrest, and the reperfusion lasted for 180 min after cross-clamp removal. The counterparts in the SO group were only subjected to median sternotomy. Histopathological analysis was used to detect mucosal injury, and Pick-and-Mix custom miRNA real-time polymerase chain reaction (PCR) panels containing 306 unique primer sets were utilized to assay unpooled intestinal samples harvested from the two groups.
RESULTS: The intestinal mucosa of the animals that were subjected to 24 °C HCA exhibited representative ischemic reperfusion injury of grade 2 or 3 according to the Chiu score. Such intestinal mucosal injuries, with the subepithelial space and epithelial layer lifting away from the lamina propria, were accompanied by shortened and irregular villi. On the contrary, the intestinal mucosa remained normal in the sham-operated animals. In total, twenty-five miRNAs were differentially expressed between the two groups (15 upregulated and 10 downregulated in the HCA group). Among these, eight miRNAs (miR-122, miR-221-5p, miR-31, miR-421-5p, miR-4333, miR-499-3p, miR-542 and let-7d-3p) were significantly dysregulated (four higher and four lower). The expression of miR-122 was significantly (5.37-fold) increased in the HCA group vs the SO group, indicating that it may play a key role in HCA-induced mucosal injury.
CONCLUSION: Exposure to HCA caused intestinal miRNA dysregulation and barrier dysfunction in swine. These altered miRNAs might be related to the protection or destruction of the intestinal barrier.
Core tip: Swine intestine was subjected to hypothermia, cardiopulmonary bypass and ischemia/reperfusion following hypothermic circulatory arrest (HCA). These factors caused barrier dysfunction, resulting in gastrointestinal complications. Histopathological and microRNA (miRNA) array analyses were used to investigate the effects of HCA on the gut barrier. HCA was found to disturb barrier function in the small intestine and influence the miRNA levels in swine. Our results contribute to the body of research examining gut barrier function following HCA in vivo.