Role of epithelial-mesenchymal transition in chemoresistance in pancreatic ductal adenocarcinoma

doi:10.12998/wjcc.v9.i19.4998

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Jul 6, 2021 (publication date) through Nov 19, 2024

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World Journal of Clinical Cases

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2307-8960

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Clin Cases. Jul 6, 2021; 9(19): 4998-5006
Published online Jul 6, 2021. doi: 10.12998/wjcc.v9.i19.4998

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Figure 1 Involvement of epithelial-mesenchymal transition in therapy resistance in pancreatic ductal adenocarcinoma. Epithelial-mesenchymal transition (EMT) is induced by various factors including signaling pathways, EMT-activating transcription factors (EMT-TFs), microRNAs, or microenvironment. Promotion of the EMT program enhances the chemoresistance in pancreatic ductal adenocarcinoma. EMT: Epithelial-mesenchymal transition; PDAC: Pancreatic ductal adenocarcinoma; EMT-TFs: Epithelial-mesenchymal transition-activating transcription factors; CAFs: Cancer-associated fibroblasts; PSCs: Pancreatic stellate cells.

Citation: Hu X, Chen W. Role of epithelial-mesenchymal transition in chemoresistance in pancreatic ductal adenocarcinoma. World J Clin Cases 2021; 9(19): 4998-5006
URL: https://www.wjgnet.com/2307-8960/full/v9/i19/4998.htm
DOI: https://dx.doi.org/10.12998/wjcc.v9.i19.4998