Research progress on venous thrombosis development in patients with malignant tumors

doi:10.12998/wjcc.v12.i11.1900

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Apr 16, 2024 (publication date) through Jul 21, 2024

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World Journal of Clinical Cases

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2307-8960

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Clin Cases. Apr 16, 2024; 12(11): 1900-1908
Published online Apr 16, 2024. doi: 10.12998/wjcc.v12.i11.1900

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Figure 1 Pathways of cancer-related thrombosis. PDAC: Pancreatic ductal adenocarcinoma; PDPN: Podoplanin; TF: Tissue factor; EV: Extracellular vesicles; VTE: Venous thromboembolism; G-CSF: Granulocyte colony-stimulating factor; MPN: Myeloproliferative-neoplasms; NET: Neutrophil extracellular trap; PAI-1: Plasminogen activator inhibitor-1.

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Figure 2 Diagram showing the mechanisms of neutrophil extracellular trap formation. MPO: Myeloperoxidase; NE: Neutrophil elastase; NADPH: Nicotinamide adenine dinucleotide phosphate; PAD: Peptidyl arginine deiminase; VWF: Von willebrand factor; NET: Neutrophil extracellular trap; ROS: Reactive oxygen species; H3cit: Histone 3 citrullination.

Citation: Wang TF, Chen Q, Deng J, Li SL, Xu Y, Ma SX. Research progress on venous thrombosis development in patients with malignant tumors. World J Clin Cases 2024; 12(11): 1900-1908
URL: https://www.wjgnet.com/2307-8960/full/v12/i11/1900.htm
DOI: https://dx.doi.org/10.12998/wjcc.v12.i11.1900