Published online Feb 16, 2024. doi: 10.12998/wjcc.v12.i5.951
Peer-review started: November 3, 2023
First decision: December 6, 2023
Revised: December 14, 2023
Accepted: January 22, 2024
Article in press: January 22, 2024
Published online: February 16, 2024
Processing time: 88 Days and 15.4 Hours
Helicobacter pylori (H. pylori) infection is a major risk factor of chronic gastritis, which perhaps influence approximately one-half of global population. H. pylori eradication is a popular treatment method for H. pylori-positive chronic gastritis, but its mechanism is far from clear.
Urinary metabolomics is gradually applied to mine the treatment mechanism of gastric diseases. However, there is no clinical study on urinary metabolomics of chronic gastritis.
This article aimed to investigate metabolic profiles of urine obtained during H. pylori eradication from patients with chronic gastritis.
In this article, we applied LC-MS-based metabolomics and network pharmacology to investigate the relationships between urinary metabolites and H. pylori-positive chronic gastritis via a clinical follow-up study.
Our study revealed the different urinary metabolic profiles of H. pylori-positive chronic gastritis before and after H. pylori eradication. The metabolites regulated by H. pylori eradication include: cis-aconitic acid, isocitric acid, citric acid, L-tyrosine, L-phenylalanine, L-tryptophan and hippuric acid, which were involved in four metabolic pathways: (1) Phenylalanine metabolism; (2) phenylalanine, tyrosine and tryptophan biosynthesis; (3) citrate cycle; and (4) glyoxylate and dicarboxylate metabolism. Integrated metabolomics and network pharmacology revealed that MPO, COMT, TPO, TH, EPX, CMA1, DDC, TPH1 and LPO were the key proteins involved in the involved the biological progress of H. pylori eradication in chronic gastritis.
Our research provides a new perspective for exploring the clinical significance of urinary metabolites in chronic gastritis.
Although this is a groundbreaking original clinical study of H. pylori-positive chronic gastritis, it is limited in that the results still require confirmation in further studies, such as targeted metabolomics, larger patient sample size, and animal experimental studies.