Published online Sep 26, 2023. doi: 10.12998/wjcc.v11.i27.6344
Peer-review started: June 24, 2023
First decision: August 9, 2023
Revised: August 18, 2023
Accepted: August 31, 2023
Article in press: August 31, 2023
Published online: September 26, 2023
Processing time: 88 Days and 4.2 Hours
Breast cancer is widely recognized as a highly malignant neoplasm in women, posing a significant risk to their overall health. The diagnostic challenges of breast cancer arise from the heterogeneity of samples and the limitations of conventional techniques. Consequently, the identification of more stable biomarkers assumes paramount importance in facilitating early breast cancer screening. In this context, bioinformatics methods have been employed to detect differentially expressed genes associated with proliferation, invasion, apoptosis, and overall survival in breast cancer.
To ascertain fundamental prognostic biomarkers in breast cancer, three databases were queried for genes associated with breast cancer as tumor markers.
Bioinformatics analysis of the molecular mechanism involved in breast cancer revealed that seven differentially expressed genes (DEGs) [calreticulin (CALR), heat shock protein family B member 1 (HSPB1), insulin-like growth Factor 1 (IGF1), interleukin-1 receptor 1 (IL1R1), Krüppel-like factor 4 (KLF4), suppressor of cytokine signaling 3 (SOCS3), and triosephosphate isomerase 1 (TPI1)] play critical roles in the progression of breast cancer. Bioinformatics were used to identify hub genes and enrichment pathways in breast cancer, illustrating a biological relationship between the pathways and gene expression in breast cancer.
Microarray data information, data processing of differentially expressed genes, protein-protein interaction network and module analysis, and survival analysis were used to mine potential biomarkers. In addition, pathway enrichment analysis was also conducted to elaborate the pathogenesis of disease.
Three Gene Expression Omnibus datasets that included breast cancer tissues and normal tissues were analyzed; 231 DEGs were identified. 7 potential biomarkers (CALR, HSPB1, IGF1, IL1R1, KLF4, SOCS3, and TPI1) were closely related to the occurrence and progression of breast cancer. The discrimination of potential markers for the model is also relatively perfect, and the discrimination for cancer group and normal group is 100%.
Through the utilization of bioinformatics analysis, the molecular mechanism of breast cancer was investigated, revealing the significant involvement of seven differentially expressed genes (CALR, HSPB1, IGF1, IL1R1, KLF4, SOCS3, and TPI1) in the advancement of breast cancer. These findings hold promise in enhancing our understanding of breast cancer pathogenesis, as well as in the identification of novel biomarkers and potential drug targets, thereby facilitating advancements in breast cancer diagnosis and therapeutics.
Bioinformatics was employed to identify hub genes and significant pathways in breast cancer, thereby establishing a biological association between the pathways and gene expression potentially implicated in the advancement of breast cancer. The utilization of bioinformatics analysis revealed the relevant genes and cellular pathways implicated in the genesis and progression of breast cancer.