Published online Apr 26, 2023. doi: 10.12998/wjcc.v11.i12.2740
Peer-review started: December 28, 2022
First decision: February 15, 2023
Revised: February 20, 2023
Accepted: March 23, 2023
Article in press: March 23, 2023
Published online: April 26, 2023
Processing time: 118 Days and 5.4 Hours
Crohn’s disease (CD) is a chronic inflammatory disorder of the gastrointestinal tract, and anemia is one of the clinical manifestations. Iron deficiency anemia (IDA) in CD is due to chronic blood loss from the ulcerated intestinal mucosa associated with diarrhea, decreased iron intake due to dietary restriction, and/or deficiency in transluminal iron absorption during the disease activity. Currently, intravenous (IV) iron replacement is the best option for the treatment of IDA in CD. Recent pharmacology progress has led ferric carboxymaltose (FCM) to correct the IDA through its pharmacokinetic characteristics.
The administration of FCM can be performed in high doses safely and well-tolerated. Given that, this study was designed to evaluate the effectiveness of FCM in the treatment of IDA in CD patients in a tertiary center in Brazil, where inflammatory bowel disease has become increasingly frequent. This clinical approach can make the quality of life of CD patients better.
The objective of this study was to analyze, through medical records, the clinical and epidemiological data of a cohort of patients with active CD who received IV FCM in the treatment of IDA to elucidate the effectiveness of the drug and compare it to iron hydroxide sucrose treatment.
It is a retrospective, observational study, which included 25 patients with active CD, severe anemia, and refractory to previous conventional treatments. Patients were evaluated two times: During previous treatment with ferric hydroxide sucrose and treatment with FCM. Epidemiological and clinical data were analyzed, besides hematimetric parameters.
The parameters of IDA assessment significantly improve after treatment with FCM. Serum hemoglobin (Hb) levels increased in 93% of patients, and in 44%, there was an increase of ≥ 2 g/dL in a single application. Moreover, 86% of the patients showed increased serum iron and ferritin and 50% in transferrin saturation. The serum iron levels at baseline showed a negative association with the ileal and colonic CD and use of biologics and a positive association with patients who developed CD later in life after the age of 40 (A3) and with a stenosing (B2) and fistulizing (B3) phenotype. The Hb and hematocrit values after ferric hydroxide sucrose treatment remained similar to those found before treatment.
FCM is an important therapeutic strategy for treating IDA in CD patients, achieving satisfactory results in refractory cases.
The study showed that FCM is an important therapeutic strategy to treat IDA in CD patients. However, there are still many gaps to be addressed in future studies about the molecular mechanism of IDA in CD. We do not yet know if IV iron replacement, besides improving the patient’s quality of life and well-being, can affect the activity of the disease and help the patient to enter clinical and endoscopic remission. Our findings support FCM as an important therapeutic strategy to treat anemia and improve the clinical status of CD patients.