Published online Jul 26, 2022. doi: 10.12998/wjcc.v10.i21.7242
Peer-review started: March 25, 2022
First decision: April 13, 2022
Revised: April 17, 2022
Accepted: June 3, 2022
Article in press: June 3, 2022
Published online: July 26, 2022
Processing time: 108 Days and 5.7 Hours
The authors have previously found in gastric cancer (GC) cell lines and animal models that the expression of hepatocyte nuclear factor 4 alpha (HNF4α) is significantly increased, and it promotes the invasion and metastasis of GC by regulating its downstream wingless-related integration site (WNT)/β-catenin signaling pathway, while negatively regulating the HNF4α/WNT signaling pathway can inhibit the development of GC.
Extending from the preclinical model to clinical GC tissues, we will continue to explore the expression of HNF4α and its downstream WNT/β-catenin signaling pathway in GC tissues, in the hope of providing new treatment ideas for inhibiting the development of GC.
To explore the expression of HNF4α, WNT5a and β-catenin in clinical GC tissues and adjacent tissues, and whether the expression of these three molecules in GC tissues is related.
Screening GC tissues containing each subtype and the adjacent para-cancerous tissues; recording the demographic characteristics of the patients and the pathological type and stage of the GC tissues; and detecting the expressions of HNF4α, WNT5a and β-catenin in the GC tissues and the para-cancerous tissues by immunohistochemistry. The expression levels of these molecules were observed under the microscope and the expression levels were recorded according to the corresponding scoring criteria. Finally, the statistical methods such as analysis of chi-square and paired chi-square were used to analyze the expression differences and correlation of HNF4α, WNT5a and β-catenin in GC tissues and para-cancerous tissues.
Compared with the para-cancerous tissues, the expressions of HNF4α (nuclear staining) and WNT5a (cytoplasmic staining) in GC tissues were significantly increased, while the expression of β-catenin (cytoplasmic staining) was significantly decreased. HNF4α expression was abundant in tubular and mucinous adenocarcinomas and relatively weak in signet ring cell carcinoma; WNT5a expression was more abundant in mucinous, than in tubular adenocarcinoma and signet ring cell carcinoma; both HNF4α and WNT5a were strongly positive in mixed GC, although no positive correlations between HNF4α and WNT5a expression in GC.
Comparing the expression of HNF4α, WNT5a and β-catenin in GC and para-cancerous tissues, we can find the expressions of HNF4α and WNT5a could serve as early diagnostic biomarkers for GC.
It is hopeful that HNF4α and WNT5a can be included into the molecules for the diagnosis of GC.