Published online May 6, 2022. doi: 10.12998/wjcc.v10.i13.4020
Peer-review started: November 2, 2021
First decision: February 14, 2022
Revised: February 25, 2022
Accepted: March 16, 2022
Article in press: March 16, 2022
Published online: May 6, 2022
Processing time: 178 Days and 18.7 Hours
Superior mesenteric artery embolism (SMAE) has acute onset and fast progression, which seriously threatens the life of patients. The early diagnosis of SMAE is related to the patients’ recovery. However, to date, there are serious challenges in the early diagnosis of SMAE, and clinical suspicion is the key to diagnosis. MDCT is one of the most important diagnostic methods for SMAE, which plays an important role in the diagnosis and prognosis of SMAE.
Superior mesenteric artery (SMA) in peripheral vessels or small-scale emboli is easy to be missed, leading to irreversible intestinal necrosis, which affects the prognosis of patients. High clinical suspicion and some extra-vascular computed tomography (CT) signs are helpful for the diagnosis of SMAE. Currently, few studies have combined clinical, biochemical and MDCT to predict the risk of irreversible intestinal necrosis and death in SMAE at early stages.
The purpose of this study is to evaluate the value of combining clinical data and MDCT in the diagnosis of SMAE and to predict the risk factors for death.
We retrospectively analyzed the clinical and MDCT data of 53 patients with SMAE confirmed by digital subtraction angiography. We analyzed the impact of a high clinical suspicion on the radiologist's diagnosis of SAME on MDCT. The patients were divided into two groups: the death and survival groups. Univariate cox regression and multivariate cox model adjusted for confounding factors were used to analyze the association trend of mortality risk with clinical and CT signs in SMAE patients.
Under the premise of high clinical suspicion of SMAE, the radiologist was able to more accurately diagnose emboli with lengths ≤ 20 mm(P = 0.014) and in areas III and IV (P = 0.024). Univariate cox regression and multivariate cox model analysis adjusted for confounding factors determined that blood lactate > 2.1 mmol/L (HR, 5.26, 95%CI: 1.04-26.69, P = 0.045) and intestinal wall thinning (HR, 9.40, 95%CI: 1.05-83.46, P = 0.044) were consistently significantly associated with mortality in SAME patients.
Increased blood lactate and intestinal wall thinning are risk factors for death in patients with SMAE. Meaningful clinical cues combined with MDCT can significantly improve the accuracy of radiologists in diagnosing SMAE with the length ≤ 20 mm and embolism in regions III and IV.
With clinical suspicion of SMAE, a multiphase enhanced CT should be performed immediately to observe the SMA trunk and peripheral vessels, as well as extravascular MDCT. Intestinal wall thinning and increased blood lactate levels might be effective predictors for death in patients with SMAE, although further validation in large sample, prospective and multicenter studies is needed. With the advent of dual-energy CT, new post-processing techniques (Iodine mapping, virtual monoenergetic imaging) may provide important information on SMA peripheral small vessel embolization and whether intestinal wall is enhanced.