Published online Oct 26, 2018. doi: 10.12998/wjcc.v6.i12.483
Peer-review started: June 6, 2018
First decision: August 1, 2018
Revised: August 23, 2018
Accepted: August 30, 2018
Article in press: August 30, 2018
Published online: October 26, 2018
Processing time: 142 Days and 23 Hours
Obesity is an important risk factor for postmenopausal breast cancer and also a poor prognostic factor among cancer patients. Moreover, obesity is associated with a number of health disorders such as insulin resistance/type-2 diabetes mellitus, hypertension, and other cardiovascular diseases. Frequently, these health disorders exhibit as components/complications of the metabolic syndrome. Nevertheless, obesity-related diseases may coexist with postmenopausal breast cancer; and these comorbid conditions could be substantial. Therefore, it may be assumed that different diseases including breast cancer could originate from a common pathological background in excessive adipose tissue. Adipocyte-released hormone-like cytokine (or adipokine) leptin behaves differently in a normal healthy state and obesity. A growing body of evidence suggests an important role of leptin in our major obesity-related health issues such as insulin resistance, hypertension, and neoplasia. In this context, this review describes the relationships of the abovementioned pathologies with leptin.
Core tip: Obesity and associated pathologies such as insulin resistance and metabolic syndrome are interrelated health disorders wherein a chronic low-grade inflammation persists. Perhaps this inflammatory condition is associated with the etiology and disease course of postmenopausal breast cancer, like other obesity-related diseases such as type-2 diabetes mellitus and hypertension. Often these diseases may coexist, and comorbidity worsens the prognosis of cancer patients. Leptin is an important adipokine (mainly released by fat cells), which may play a crucial role in these obesity-related diseases.