Published online Nov 16, 2024. doi: 10.12998/wjcc.v12.i32.6538
Revised: July 25, 2024
Accepted: July 31, 2024
Published online: November 16, 2024
Processing time: 104 Days and 12.5 Hours
In this editorial, we comment on the article by Lei et al, with a specific focus on the timing of the initiation of the antifibrotic agent pirfenidone (PFD) in the management of idiopathic pulmonary fibrosis (IPF) and its impact on lung function of IPF patients. PFD is an antifibrotic agent that is widely used in the management of IPF in both early and advanced stages. It inhibits various pathways and has antifibrotic, anti-inflammatory, and antioxidant properties. Despite dosage lowering, PFD slowed IPF progression and maintained functional capacity. The 6-min walk distance test indicated that patients tolerated adverse events well, and PFD significantly reduced the incidence of progression episodes and death. Even when a single disease-progression event occurred, continuing PFD treatment had benefits.
Core Tip: Pirfenidone is commonly used to treat idiopathic pulmonary fibrosis (IPF) in its early stages, but it also demonstrated a notable impact on the disease in its advanced stages, having a similar degree of efficacy in individuals with IPF and substantial deterioration of their lung function. It was safe when given to patients with advanced IPF and had a favorable benefit-risk profile, making it an appropriate treatment option for patients with IPF.