Published online Jul 6, 2022. doi: 10.12998/wjcc.v10.i19.6360
Peer-review started: January 23, 2022
First decision: March 23, 2022
Revised: April 1, 2022
Accepted: May 12, 2022
Article in press: May 12, 2022
Published online: July 6, 2022
Processing time: 152 Days and 5.2 Hours
Non-small-cell lung cancer (NSCLC) causes significant mortality worldwide. Patients with chronic renal failure have an increased risk of developing lung cancer. NSCLC Patients with chronic renal failure undergoing hemodialysis (HD) often exhibit poor performance, and chemotherapy is generally contraindicated. Oral epidermal growth factor receptor (EGFR)-tyrosine kinase inhibitors (TKIs) are effective treatment agents for NSCLC patients. However, the benefits and adverse effects of EGFR-TKIs in NSCLC undergoing HD are known. There are no clinical studies on the effects of EGFR-TKIs on NSCLC patients undergoing HD. We reviewed all previous case reports about EGFR-TKIs in NSCLC patients undergoing HD. It is difficult to design studies about the effects of EGFR-TKIs in patients undergoing HD, and this review is quite important. EGFR-TKIs are well tolerated in patients undergoing HD. The main routes of elimination of EGFR-TKIs are metabolism via the liver, and renal elimination is minor. The recommended doses and pharmacokinetics of these EGFR-TKIs for patients undergoing HD are similar to those for patients with normal renal function. The plasma protein binding of EGFR-TKIs is very high, and it is not necessary to adjust the dose after HD. In conclusion, EGFR-TKIs are effective and well tolerated in patients undergoing HD.
Core Tip: Lung cancer causes significant mortality worldwide. Patients with chronic renal failure increase the difficulty of treatment. Oral epidermal growth factor receptor (EGFR)-tyrosine kinase inhibitors (TKIs) are effective treatment agents for non-small-cell lung cancer (NSCLC) patients. According to this review, EGFR-TKIs are suitable for treating NSCLC patients undergoing hemodialysis (HD) owing to their good response rates and tolerance. The main routes of elimination of EGFR-TKIs are via the liver and excretion via feces. The recommended doses and pharmacokinetics of EGFR-TKIs for patients undergoing HD are similar to those with normal renal function. The plasma protein binding of EGFR-TKIs is high, and it is not necessary to adjust the dose after HD.