Copyright ©The Author(s) 2022.
World J Gastrointest Oncol. Jan 15, 2022; 14(1): 90-109
Published online Jan 15, 2022. doi: 10.4251/wjgo.v14.i1.90
Figure 1
Figure 1 Histone modifications in gastric cancer. Layers show different histone modifications. Blue panel: Modification types; Pink panel: Modified residues and catalytic enzymes; Green panel: Epigenetic alterations sites; Yellow panel: Biological functions regulated by histone modifications. HAT; Histone acetylase; HDAC: Histone deacetylase; HMT: Histone methyltransferase; HDM: Histone demethylase; UBE: Ubiquitin enzyme; DUB: Deubiquitinase.
Figure 2
Figure 2 DNA methylation in gastric cancer. Aberrant methylation in promoter, shore area and gene body altered gene expression and involves in gastric carcinogenesis.
Figure 3
Figure 3 Non coding RNA in gastric cancer. The major mechanism and biological function of lncRNA, miRNA and circRNA in gastric cancer.