Published online May 15, 2024. doi: 10.4251/wjgo.v16.i5.2113
Peer-review started: January 2, 2024
First decision: January 10, 2024
Revised: January 19, 2024
Accepted: March 7, 2024
Article in press: March 7, 2024
Published online: May 15, 2024
Processing time: 128 Days and 3.1 Hours
Mesenchymal stem cells have emerged as viable and ideal therapeutic agents demonstrating efficacy in immunomodulation, antiviral activity, and tissue regeneration/repair after injury, as evidenced by preclinical studies and clinical trials. The ongoing coronavirus disease 2019 (COVID-19) pandemic, fueled by the highly infectious severe acute respiratory syndrome coronavirus 2, has prompted the exploration of potential treatments. Accumulating evidence supports the effectiveness of adipose tissue-derived mesenchymal stem cells (ADSCs) as an effective therapeutic approach for managing COVID-19. However, the underlying immunomodulatory effects on the mRNA expression of Th cell-related transcription factors (TFs) as well as cytokine release in peripheral blood mononuclear cells (PBMCs) need to be elu
This study investigated the effect of ADSC therapy on the mRNA expression of TFs and cytokine release in colorectal cancer (CRC) patients with severe COVID-19 (CRC+ patients).
This study investigated the immunomodulatory effect of ADSCs in CRC+ patients.
PBMCs from CRC+ patients (PBMCs-C+) and age-matched CRC patients (PBMCs-C) were stimulated and cultured in the presence/absence of ADSC. The mRNA levels of T-box TF TBX21 (T-bet), GATA binding protein 3 (GATA-3), RAR-related orphan receptor C (RORC), and forkhead box P3 (FoxP3) in the PBMCs were determined by real-time reverse transcriptase-polymerase chain reaction; the culture supernatants were examined to measure the levels of interferon gamma (IFN-γ), interleukin 4 (IL-4), IL-17A, and transforming growth factor beta one (TGF-β1) using an enzyme-linked immunosorbent assay.
Compared with the PBMCs-C group, the mRNA levels of TFs (T-bet and RORC) and the release of cytokines (IFN-γ and IL-17A) were higher in the PBMCs-C+group. Additionally, a significant decrease in FoxP3 mRNA and TGF-β1 Levels, coupled with an increase in T-bet/GATA-3, RORC/FoxP3, IFN-γ/IL-4, and IL-17A/TGF-β1 ratios were observed in the PBMCs-C+group. Further analysis revealed that ADSC therapy significantly induced functional regulatory T cell (Treg) subsets, as evidenced by an increase in the levels of FoxP3 mRNA and TGF-β1, and a decrease in the mRNA levels of TFs (T-bet and RORC), release of cytokines (IFN-γ and IL-17A), and T-bet/GATA-3, RORC/FoxP3, IFN-γ/IL-4, and IL-17A/TGF-β1 ratios, compared with the PBMCs-C+alone group.
ADSCs contributed to the immunosuppressive effects on PBMCs-C+.
Our study demonstrated that ADSC intervention, as a living immunomodulatory agent, is a promising therapeutic approach to create an anti-inflammatory environment by mitigating excessive inflammatory reactions and promoting Treg-biased tolerance immune responses. Thus, ADSCs are expected to be a viable alternative treatment option for preventing severe COVID-19 progression.