Published online Apr 15, 2024. doi: 10.4251/wjgo.v16.i4.1578
Peer-review started: October 8, 2023
First decision: December 15, 2023
Revised: December 24, 2023
Accepted: January 23, 2024
Article in press: January 23, 2024
Published online: April 15, 2024
Processing time: 186 Days and 1 Hours
The association between heat shock proteins (HSPs) and cancer prognosis has generated significant interest, offering potential implications for clinical decision-making in cancer management. HSPs and their regulatory factors, such as heat shock factor (HSF)1 and CHIP, play an important role in cellular protection against stress events, and are overexpressed in some types of cancer.
The prognostic significance of HSPs and their regulatory factors, such as HSF1 and CHIP, are poorly understood in esophageal and esophagogastric cancer.
We conducted a systematic review and meta-analysis to investigate the relationship between HSP expression and prognosis in esophageal and esophagogastric cancer.
A systematic review was conducted in accordance with PRISMA recommendations, on Embase, PubMed, Cochrane, and LILACS. Cohort, case-control, and cross-sectional studies of patients with esophagus or esophagogastric cancer were included. HSP-positive patients were compared with HSP-negative, and the endpoints analyzed were lymph node metastasis, tumor depth, distant metastasis, and overall survival (OS). HSPs were stratified according to the HSP family, and the summary risk difference (RD) was calculated using a random-effect model.
The final selection comprised 27 studies, including esophageal squamous cell carcinoma (21), esophagogastric adenocarcinoma (5), and mixed neoplasms (1). The pooled sample size was 3465 patients. HSP40 and 60 were associated with a higher 3-year OS, while HSF1 was associated with a poor 3-year OS. The other HSP families were not associated with long-term survival. HSF1 was associated with a higher probability of lymph node metastasis. HSP40 was associated with a lower probability of lymph node dissemination. The expression of other HSP families was not significantly related to tumor depth and lymph node or distant metastasis.
Our findings demonstrated that the expression levels of some families of HSP, such as HSP40 and 60 and HSF1, are associated with long-term survival and lymph node dissemination in esophageal and esophagogastric cancer.
The results of this study underscore the noteworthy prognostic implications of HSPs within the realm of cancer research, suggesting potential avenues for therapeutic interventions. The ongoing exploration of this field offers the prospect of furthering precision medicine and developing targeted strategies for the management of esophageal and esophagogastric cancer.