Published online Mar 15, 2024. doi: 10.4251/wjgo.v16.i3.670
Peer-review started: November 7, 2023
First decision: December 31, 2023
Revised: January 16, 2024
Accepted: February 4, 2024
Article in press: February 4, 2024
Published online: March 15, 2024
Processing time: 125 Days and 20.6 Hours
The morbidity and mortality of colorectal cancer (CRC) are among the highest in the world. When the balance between pigment epithelium-derived factor (PEDF), which inhibits angiogenesis, and vascular endothelial growth factor (VEGF), which stimulates angiogenesis, is broken, it can lead to uncontrolled angiogenesis and promote the occurrence of tumors. Therefore, it is necessary to find more therapeutic targets for early intervention and late treatment of CRC.
The safety and efficacy of targeted drugs targeting VEGF in the treatment of CRC have been confirmed and promoted. PEDF is the anti-VEGF factor. At present, no toxicity caused by PEDF preparation itself has been observed in anti-tumor vascular animal models. It is worth exploring the possibility of PEDF as a new target for early prevention and late treatment of CRC.
Study of the expression and significance of PEDF, VEGF, and CD31-stained microvessel density values (CD31-MVD) in normal colorectal mucosa, adenoma, and CRC.
We collected 50 cases of normal intestinal mucosa, 50 cases of colorectal adenoma and 50 cases of colon cancer as normal control group, adenoma group and CRC group, respectively. Immunohistochemical staining was used to detect the expression of PEDF and VEGF in the three groups, and the differences were analyzed. The relationship between the expression of PEDF and VEGF and the clinicopathological factors of CRC was studied. CD31-MVD was recorded in the three groups, and the correlation between PEDF, VEGF and CD31-MVD in colorectal adenoma group and CRC group was analyzed.
The positive expression rate and expression intensity of PEDF in normal control group, adenoma group and CRC group gradually decreased, while that of VEGF gradually increased. In the CRC group, the positive expression rate of PEDF decreased with the increase of differentiation degree, invasion depth, lymph node metastasis, distant metastasis and TNM stage. The opposite was observed for VEGF high expression. In the colorectal adenoma group, the expression intensity of PEDF was negatively correlated with CD31-MVD, but there was no significant difference in VEGF expression. PEDF expression was negatively correlated with CD31-MVD and VEGF expression in CRC group. The expression of VEGF was positively correlated with CD31-MVD.
It is possible that PEDF can be used as a new treatment and prevention target for CRC by upregulating the expression of PEDF while inhibiting the expression of VEGF.
We will further expand our sample size to equalize the proportion of various types of adenomas in the colorectal adenoma group and the proportion of various pathological types of CRC in the CRC group to further confirm our conclusion.