Published online Feb 15, 2024. doi: 10.4251/wjgo.v16.i2.436
Peer-review started: August 19, 2023
First decision: December 5, 2023
Revised: December 13, 2023
Accepted: January 9, 2024
Article in press: January 9, 2024
Published online: February 15, 2024
Processing time: 166 Days and 17.8 Hours
The global pandemic of coronavirus disease 2019 (COVID-19) posed an enormous crisis challenge to the public health system, and the negative effects continue. Numerous clinical examples have shown that COVID-19 appears to have a significant impact on the treatment of patients with liver cancer compared to the normal population, and that the prevalence of COVID-19 infection is significantly higher in patients with liver cancer.
Both COVID-19 and liver cancer deserve serious attention in an attempt to gain a deeper understanding of their potential association, and at the same time, there is a lack of joint analyses and explorations of these two diseases.
Based on the bioinformatics approach to explore the potential link between COVID-19 and hepatocellular carcinoma (HCC), it is expected that the common regulatory mechanism of both will be discovered.
The datasets of COVID-19 and liver cancer were obtained from the Gene Expression Omnibus database. The common differentially expressed genes (DGEs) of COVID-19 and HCC were obtained by R software, and they were subjected to Gene Ontology/Kyoto Encyclopedia of Genes and Genomes enrichment analysis, protein-protein interaction network construction, and hub genes were screened and enriched from DGEs. Subsequently, the differential expression of hub genes in diseases was verified, and the regulatory networks of transcription factors and hub genes were constructed. The expression of some hub genes in liver cancer was verified by quantitative reverse transcriptase polymerase chain reaction.
Five hundred and eighteen common differentially expressed genes were obtained by screening, from which the 15 most critical hub genes were identified. Functional enrichment analysis of the pivotal genes showed that these hub genes were closely related to P53 signalling pathway regulation, cell cycle and other functions, and might serve as potential diagnostic markers for COVID-19 and liver cancer. Finally, some of these hub genes were selected for in vitro expression validation in liver cancer cells.
Our findings suggest that the common pathogenic mechanism of COVID-19 and liver cancer may be mediated by pathways such as the role of specific hub genes and the P53 signalling pathway.
The construction of a dual-disease model of COVID-19 and liver cancer is of great interest for further validation of the correlation between the two pathogenic mechanisms.