Published online Mar 15, 2023. doi: 10.4251/wjgo.v15.i3.523
Peer-review started: November 11, 2022
First decision: November 28, 2022
Revised: December 8, 2022
Accepted: February 10, 2023
Article in press: February 10, 2023
Published online: March 15, 2023
Processing time: 123 Days and 10.2 Hours
Studies have shown an increased risk for various malignancies, particularly lymphomas, small intestinal adenocarcinoma, and other gastrointestinal malignancies, in celiac disease (CD) patients. However, the magnitude of the risk of pancreatic cancer (PC) in association with CD is much less clear. Nevertheless, despite the magnitude of the risk remaining debatable, the unequivocal association between CD and PC remains.
Although malignancy occurring in the setting of CD has been well recognized; however, there is considerable, but not definitive, evidence that strict compliance to CD is associated with increased risk for the development of PC. Furthermore, owing to the lack of control for known risk factors of PC, any independent associations between these diseases cannot be assessed based on previous studies; given the high incidence of CD and the poor outcomes associated with PC, any such potential association warrants further investigation.
This study aimed to assess the risk of PC in patients with CD.
A population-based, multicenter, propensity score-matched cohort study included 155877 patients with CD and 234103 patients without CD (non-CD, controls). To reduce confounding effects, we performed a 1:1 propensity score matching with each patient in the main group to a patient in the control group. The incidence of PC was estimated using a Cox proportional hazards model with a hazard ratio (HR) and 95% confidence interval (CI).
During the follow-up, 309 patients with CD developed PC, whereas 240 patients developed PC in the control group (HR = 1.29; 95%CI: 1.09-1.53). In the secondary analyses in the first year after diagnosis of CD, patients with CD were at a significant increase in risk for PC; 151 patients with CD had an incidence of PC compared with 96 incidences of PC among the patients in the non-CD control group (HR = 1.56; 95%CI: 1.20-2.01) and sensitivity analysis showed similar magnitude to the one generated in the primary and secondary analysis.
This multicenter, propensity score-matched cohort study reveals that patients with CD are at increased risk of PC. Risk elevation persists beyond the first year after diagnosis to reference individuals without CD from the general population.
We still know little about the risk factors and mechanisms contributing to developing malignancies among individuals. Further experimental data and long-term follow-up studies are required to elucidate the pathogenic mechanisms and to ensure better comprehension of the association between PC and CD.