Published online Nov 15, 2019. doi: 10.4251/wjgo.v11.i11.983
Peer-review started: March 28, 2019
First decision: April 15, 2019
Revised: July 26, 2019
Accepted: September 12, 2019
Article in press: September 12, 2019
Published online: November 15, 2019
Processing time: 233 Days and 22.6 Hours
Hepatocellular carcinoma (HCC), one of the most frequent liver cancer subtype, has posed a serious health issue in the world. Hepatitis virus is recognized to be a major factor leading to HCC. Recently, a verity of genetic markers as well as prediction models are proposed to improve HCC treatment. At the same time, numerous statistical techniques are also utilized to mine data in numerous large-scale public databases. Thanks to the well-developed clinical approaches, prognosis models with a higher accuracy and robustness can be established for HCC.
This study aimed to establish a prognosis model on the basis of HCC molecular biomarkers. Typically, long non-coding RNAs (lncRNAs) have been recognized as new predictive factors. Great efforts have been made to establish lncRNA-based HCC models, however, the lncRNA features for hepatitis virus positive HCC (VHCC) are not available at present.
This study aimed to develop a prognostic lncRNA feature for VHCC based on candidate lncRNAs through analyzing data collected from The Cancer Genome Atlas (TCGA) database.
Specifically, the least absolute shrinkage and selection operator (LASSO), the most advanced statistic algorithm, was used to establish the prediction model. This approach was carried out on the basis of typical lncRNAs selected according to the expression profiles of lncRNAs collected from the TCGA database. The as-established lncRNA feature was validated, and its clinical applicability was also examined.
Using LASSO, a risk score system was established to predict the overall survival (OS) for VHCC, which incorporated eight lncRNAs (including AC005722.2, AC107959.3, AL353803.1, AL589182.1, AP000844.2, AP002478.1, FLJ36000, and NPSR1-AS1). Notably, the as-constructed lncRNA feature was helpful in stratifying the risk for VHCC. To extend the model application range, a nomogram was also constructed, which involved both the lncRNA feature and other clinical features. Our results also suggested that the lncRNAs were markedly enriched in the Wnt signaling pathway, angiogenesis, the p53 pathway, and the PI3 kinase pathway.
The as-constructed signature based on eight lncRNAs displayed a favorable capacity in predicting the prognosis for VHCC patients, which can also contribute to risk stratification and may provide more useful clinical advice for individual patients.
Our results further support that lncRNAs can serve as possible functional regulating factors for the progression of VHCC. It is the future direction to search for the effective molecular biomarkers and predictive factors for the prognosis of HCC patients.