Published online Nov 15, 2018. doi: 10.4251/wjgo.v10.i11.421
Peer-review started: August 7, 2018
First decision: August 31, 2018
Revised: September 7, 2018
Accepted: October 10, 2018
Article in press: October 10, 2018
Published online: November 15, 2018
Processing time: 101 Days and 12.5 Hours
Although FOLFIRINOX is one of the universally-used chemotherapies for pancreatic cancer, its relatively high rate of adverse events is still a major concern. Several studies suggest that dose-modified FOLFIRINOX (mFOLFIRINOX) can improve safety with comparable efficacy compared to the standard FOLFIRINOX (sFOLFIRINOX). However, clinical feasibility and the optimal strategy of mFOLFIRINOX remains unclear.
Previous studies on mFOLFIRINOX made conclusions based on comparing their results to the results of historical phase III trials of FOLFIRINOX. To date, direct comparative studies between sFOLFIRINOX and mFOLFIRINOX for pancreatic cancer is lacking.
We directly compared the safety and efficacy of sFOLFIRINOX and mFOLFIRINOX in a single study. This could help clarify the clinical applicability of mFOLFIRINOX.
The medical records of 130 pancreatic cancer patients [sFOLFIRINOX (n = 88), mFOLFIRINOX (n = 42)] were retrospectively reviewed. The objective response rate (ORR), disease control rate (DCR), progression-free survival (PFS), and overall survival (OS) were compared for efficacy analysis. Severe (≥ grade three) adverse event (AE) rates of the two groups were compared for toxicity analysis.
Although the median relative dose intensities of each of the drugs were significantly lower in the mFOLFIRINOX group, the response rates and survival were not different between the two groups (ORR: 39.8% vs 35.7%, P = 0.656; DCR: 80.7% vs 83.3%, P = 0.716; PFS: 8.7 mo vs 8.1 mo, P = 0.272; OS: 13.9 mo vs 13.7 mo, P = 0.476). Severe AE rates, including neutropenia (83.0% vs 66.7%; P = 0.044), anorexia (48.9% vs 28.6%; P = 0.029), and diarrhea (13.6% vs 0.0%; P = 0.009), were significantly lower in the mFOLFIRINOX group.
In this direct comparative restrospective study, mFOLFIRINOX showed comparable efficacy to sFOLFIRINOX, with a better toxicity profile. Given the relatively high toxicity of sFOLFIRINOX, initiating FOLFIRINOX treatment, if clinically required, with 75% of the standard-dose could be an appropriate option to reduce toxicity concerns without compromising efficacy.
In the future, prospective comparative studies need to be conducted to determine the optimal dose modification of FOLFIRINOX and who will benefit from this strategy.