Published online Nov 15, 2018. doi: 10.4251/wjgo.v10.i11.367
Peer-review started: July 3, 2018
First decision: July 19, 2018
Revised: August 1, 2018
Accepted: October 9, 2018
Article in press: October 9, 2018
Published online: November 15, 2018
Processing time: 135 Days and 20.4 Hours
Autophagy is a basic catabolic process closely associated with degradation of cellular components. The role of autophagy in colorectal cancer (CRC) remains controversial. The mechanism of autophagy has been identified as protecting mechanism against tumorigenesis by isolation of damaged organelles or as cytoprotective provides energy in hypoxic regions of CRC tumors. Mutations in proto-oncogenes, such as RAS and BRAF, have been associated with autophagy initiation through signaling pathways of BRAF/MEK/ERK and PI3K/AKT/mTOR. A combination therapy of chemotherapeutic agents and autophagy inhibitors such as hydroxychloroquine or immunotherapy might represent a major step that could be evaluated as a putative novel therapeutic strategy in CRC patients.
Core tip: The significant role of autophagy in maintaining the balance of tumorigenesis and cancer cell death remains controversial. The last decade grown body of evidence support the notion that autophagy is a promising target for many malignant tumors, including colorectal cancer (CRC). A novel therapeutic approach which could involve autophagy inhibitors or immunotherapy plus chemotherapeutic drugs could open a new field for treating patients with CRC.