Metabolic and immune links between sarcopenia and liver disease

doi:10.4254/wjh.v17.i8.109444

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Aug 27, 2025 (publication date) through Aug 30, 2025

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World Journal of Hepatology

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1948-5182

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Hepatol. Aug 27, 2025; 17(8): 109444
Published online Aug 27, 2025. doi: 10.4254/wjh.v17.i8.109444

Metabolic and immune links between sarcopenia and liver disease

Stanislav Nikolaevich Kotlyarov

Stanislav Nikolaevich Kotlyarov, Department of Nurse, Ryazan State Medical University, Ryazan 390005, Russia

Author contributions: Kotlyarov SN contributed to conceptualization, methodology, validation, resources, data curation, writing original draft preparation, review and editing, supervision, and project administration.

Conflict-of-interest statement: The author reports no relevant conflicts of interest for this article.

Open Access: This article is an open-access article that was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution NonCommercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: https://creativecommons.org/Licenses/by-nc/4.0/

Corresponding author: Stanislav Nikolaevich Kotlyarov, PhD, Professor, Department of Nurse, Ryazan State Medical University, Russian Federation, Vysokovoltnaya 9, Ryazan 390005, Russia. skmr1@yandex.ru

Received: May 12, 2025
Revised: May 25, 2025
Accepted: July 24, 2025
Published online: August 27, 2025
Processing time: 108 Days and 21.2 Hours

Core Tip

Core Tip: Sarcopenia is characterized by loss of mass, strength, and skeletal muscle function. Skeletal muscle has numerous direct and indirect metabolic and immune links with the liver, which are disrupted in diseases such as metabolic dysfunction-associated steatotic liver disease and cirrhosis. Disruption of these links contributes to the premature development of sarcopenia, which further worsens the course of these diseases and the prognosis.