Published online Aug 28, 2023. doi: 10.3748/wjg.v29.i32.4900
Peer-review started: June 4, 2023
First decision: July 8, 2023
Revised: July 20, 2023
Accepted: July 31, 2023
Article in press: July 31, 2023
Published online: August 28, 2023
Processing time: 81 Days and 14.7 Hours
Although hepatitis B virus (HBV) infection is the most common cause of liver disease in China, the occurrence of HBV infection in Wilson disease (WD) patients and the clinical manifestations of concurrent WD and HBV infections have rarely been reported.
Our study suggests that both WD and HBV infections may coexist. The clinical symptoms of concurrent WD and HBV infections are difficult to distinguish from those of simple viral hepatitis. Therefore, the existence of WD may be hidden. The study of concurrent WD and HBV infection deserves careful consideration.
To investigate the incidence of HBV infection in patients with WD and to analyse how HBV infection affects WD.
The clinical data of patients with WD were analyzed retrospectively, and the data of patients with concurrent WD and HBV infection were compared with those of patients with isolated WD. Considering the rarity of the disease, the sample size of the WD patient cohort was very large.
Among a total of 915 WD patients recruited, the total prevalence of current and previous HBV infection was 2.1% and 9.2% respectively. The main finding of this study was the identification of 19 patients with concurrent WD and chronic hepatitis B (CHB) infection. The mean delay in the diagnosis of WD in patients with concurrent WD and CHB infection was 32.5 mo, which was significantly longer than that in patients with isolated WD (10.5 mo). The rates of severe liver disease and mortality in patients with concurrent WD and CHB infection were significantly higher than those in patients with isolated WD (63.1% vs 19.3%), respectively. Binary logistic regression analysis revealed a significantly higher risk of severe liver disease at the diagnosis of WD in patients with current HBV infection or previous HBV infection than in patients with isolated WD.
Our study indicates that the prevalence of HBV infection stratified by sex and age in patients with WD is similar to that in the general population. There was a significant delay in the diagnosis of WD in CHB patients. HBV infection is an independent risk factor for severe liver disease in WD patients. WD should be considered and excluded in some patients with CHB infection.
As we found that previous HBV infection was an independent factor in the exacerbation of WD, the mechanism of which is speculative, future studies could further explore the mechanism by which WD is exacerbated by previous HBV infection and whether it is related to occult HBV infection.