Published online Feb 7, 2021. doi: 10.3748/wjg.v27.i5.391
Peer-review started: October 12, 2020
First decision: November 23, 2020
Revised: November 30, 2020
Accepted: December 17, 2020
Article in press: December 17, 2020
Published online: February 7, 2021
Processing time: 108 Days and 15.5 Hours
Post-cholecystectomy diarrhea (PCD) frequently occurs in patients following gallbladder removal. PCD is difficult to treat. After cholecystectomy, bile enters the duodenum directly, independent of the timing of meals. The interaction between the bile acids and the intestinal microbes is changed. Therefore, the occurrence of PCD may be related to the change in microbiota.
Little is known regarding the intestinal microbiota characteristics in PCD patients. High-throughput sequencing of the 16S rRNA gene can be used to characterize the composition and diversity of the complex intestinal microbial community.
To identify changes in the intestinal microbiota and to better understand the role of the intestinal microbiota in PCD patients.
The diversity and profiles of the gut microbiota were analyzed by performing high-through put 16S rRNA gene sequencing. The gut microbiota in a healthy control (HC) group and a post-cholecystectomy (PC) group were characterized. Subsequently, the PC group was further divided into a PCD group and a post-cholecystectomy non-diarrhea group (PCND) according to the patients’ clinical symptoms. The composition, diversity and richness of microbial communities were detected and compared.
The PC group had fewer OTUs than the HC group. β-diversity indicated a decreased microbial diversity in the PC group. Fifteen taxa with differential abundance between the HC and PC groups were identified. In the PCD group compared to the PCND group, significant decreases in microbial diversity, Firmicutes/Bacteroidetes ratio, and richness of probiotic microbiota (Bifidobacterium and Lactococcus), and an increase in detrimental microbiota (Prevotella and Sutterella) were observed. Moreover, a negative correlation was observed between Prevotella and Bifidobacterium. By performing Kyoto Encyclopedia of Genes and Genomes functional analysis, we found that the abundances of gut microbiota involved in lipid metabolism pathways were markedly lower in the PCD group compared to the PCND group.
These findings demonstrate the association between PCD and the gut microbiota. Gut dysbiosis in PCD patients is a result of differences in microbial structure, diversity, and abundance when compared to PCND patients. Decreased diversity and abundance of the microbial community in PC patients can cause diarrhea.
This study demonstrated that gut dysbiosis may play a critical role in PCD, especially regarding Prevotella and Bifidobacterium. Thus, this study provides new insights into potential therapeutics that could target the microbiota to attenuate PCD.