Published online Feb 28, 2019. doi: 10.3748/wjg.v25.i8.1012
Peer-review started: December 6, 2018
First decision: January 6, 2019
Revised: January 18, 2019
Accepted: January 26, 2019
Article in press: January 26, 2019
Published online: February 28, 2019
Processing time: 85 Days and 13.6 Hours
Gastric cancer (GC) is one of the most common malignancies worldwide. Despite the advances in diagnosis and treatment of GC in recent decades, prognosis of GC patients is still poor. It is of great importance to identify biomarkers that could be helpful in the improvement of screening of high-risk individuals, early diagnosis, and predicting outcome for the individualized therapy.
The microRNA lethal-7 (let-7) often exhibits tumor-suppressor functions in tumorigenesis. Single nucleotide polymorphisms (SNPs) in the let-7 gene region or let-7 target genes have been reported to modulate the risk of several cancers including breast cancer and lung cancer. In GC, the related studies are limited
By including a relatively large number of GC patients, this study aimed to determine the role of let-7-related polymorphisms in GC in a Chinese population.
From 2008 to 2013, 898 consecutive GC patients and 992 tumor-free controls were recruited into the study. GC patients were followed periodically to determine their prognosis. Ten SNPs in the let-7 gene region or their target mRNAs were genotyped using MassArray system and the associations with the risk or overall survival of GC were analyzed.
Two SNPs in let-7 target genes were associated with GC in a dose-dependent manner. Rs3811463 in the 3’-UTR of LIN28A was associated with lower risk of GC and the risk was reduced by 26% with each increase of the C allele of rs3811463. The rs10889677 in the 3’-UTR of IL23R was corresponded to the prognosis of GC, and the death risk increased by 25% with each increment of the C allele of rs10889677, after controlling for clinicopathological parameters.
Let-7-related SNPs were related to GC. The rs3811463 in LIN28A is associated with the susceptibility to and rs10889677 in IL23R is associated with the prognosis of GC.
Our research adds evidence that polymorphisms represent a genetic factor to modify the susceptibility to and prognosis of GC. The underlying mechanisms of the associations should be elucidated in future studies.