Published online Nov 21, 2019. doi: 10.3748/wjg.v25.i43.6465
Peer-review started: July 25, 2019
First decision: August 27, 2019
Revised: October 24, 2019
Accepted: November 7, 2019
Article in press: November 7, 2019
Published online: November 21, 2019
Processing time: 118 Days and 18.9 Hours
Ulcerative colitis (UC) is a chronic disease associated with many other diseases such as rheumatoid arthritis, multiple sclerosis, lupus, psoriasis, hypothyroidism, and metabolic syndrome (MetS). Among these diseases, the MetS is the most common comorbidity. There is no evidence considering whether the comorbidity with MetS alters the course of the UC.
We hope to offer reliable evidence that MetS affects the outcome of the UC, given the increasingly common comorbidity.
Test the impact of the MetS on the severity of UC and the local and systemic immune response.
A total of 89 patients with de novo confirmed UC were enrolled in this cross-sectional study, and they were further divided in two groups, according to ATP III criteria: group without MetS (no MetS) and group with MetS. Severity of UC was determined by histological and clinical scores, fecal and serum cytokines levels were determined using an enzyme-linked immunosorbent assay, while cellular makeup of colon infiltrations was determined by flow cytometry.
Research resultsWhen compared to UC patients without MetS, clinically and histologically milder disease with higher serum level of immunosuppressive cytokine interleukin- 10 (IL-10) and fecal content of Galectin-3 (Gal-3) was observed in subjects with UC and MetS. This was accompanied with predomination of IL-10 over pro-inflammatory cytokines tumor necrosis factor α (TNF-α), interleukin-6, and interleukin-17 (IL-17) in the sera as well as Gal-3 over TNF-α and IL-17 in feces of UC patients with MetS. Significantly lower systemic values of IL-17, higher values of IL-10 and Gal-3 values in feces were determined in MetS patients in especially same clinical, endoscopic and histopathological stage of UC as patients without MetS. In addition, UC + MetS patients had higher percentage of IL-10 producing and Gal-3 expressing innate and acquired immune cells in lamina propria of affected colon tissue.
UC patients with MetS have clinically and histologically milder disease. Predominance of Gal-3 and IL-10 over pro-inflammatory mediators in patients with MetS may present a mechanism for limiting the inflammatory process and subsequent tissue damage in UC.
Future studies are needed to investigate the exact mechanism underlying the protective effect of MetS in biology of UC. And it is necessary to determinate the influence of developmental stages of MetS on the severity of UC. Large sample size studies are also required to confirm the current findings.