Colorectal Cancer
Copyright ©The Author(s) 2003. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Nov 15, 2003; 9(11): 2469-2473
Published online Nov 15, 2003. doi: 10.3748/wjg.v9.i11.2469
Lack of inhibitory effects of Lactic acid bacteria on 1,2-dimethylhydrazine-induced colon tumors in rats
Wei Li, Chong-Bi Li
Wei Li, Department of Obstetrics and Gynecology, First People’s Hospital of Hangzhou, Hangzhou 310006, Zhejiang Province, China
Chong-Bi Li, Department of Biology, College of Zhaoqing, Duanzhou-Qu, Zhaoqing 526000, Guangdong Province, China
Author contributions: All authors contributed equally to the work.
Correspondence to: Dr. Wei Li, Department of Obstetrics and Gynecology, First People’s Hospital of Hangzhou, 261 Wansha-lu, Shangcheng-Qu, Hangzhou 310006, Zhejiang Province, China. lwmyj@hotmail.com
Telephone: +86-571-87065701-4331 Fax: +86-571-87914773
Received: March 5, 2003
Revised: March 25, 2003
Accepted: April 1, 2003
Published online: November 15, 2003
Abstract

AIM: A myriad of healthful effects has been attributed to the probiotic lactic acid bacteria, perhaps the most controversial issue remains that of anticancer activity. This study was aimed at investigating the putative anti-cancer effects of lactic acid bacteria strains on the progression of colon tumor in 1,2-dimethylhydrazine (DMH)-treated animals.

METHODS: The strain of lactic acid bacteria used in this study was lactic acid bacteria NZ9000 that conformed to the characteristics of plasmid free. Sixty male Wistar rats were given subcutaneous injections of DMH at a dose of 40 mg/kg body wt or saline once a week for 10 wk. The rats were divided into 6 experimental groups. After the last DMH injection, animals in groups 1 and 4 were gavaged with 1 mL of lactic acid bacteria at a dose of 5 × 109 per day or vehicle until sacrifice at the end of week 22 or week 52. Animals in groups 1-3 were killed at the end of week 22 for histopathological examination. The whole period of experimental observation was 52 wk.

RESULTS: By the end of 22nd week, final average body weights of the rats treated with DMH alone and all animals receiving lactic acid bacteria were significantly decreased compared with the vehicle control (P < 0.05). No differences in tumor incidence, multiplicity, dimensions and stage in the colonic mucosa were observed among the groups. At week 52, the survival rate of the rats administered lactic acid bacteria was lower than that of the rats treated with DMH that were fed on control fluids of non-lactococcus lactis. The mean survival time of lactic acid bacteria-treated animals was 39 wk.

CONCLUSION: These results indicate that lactic acid bacteria lacks inhibitory effects on the progression of colon tumor in DMH-treated animals, and does not support the hypothesis that alteration of colonic flora may exert an influence on the progression of colon tumor.

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