Original Articles
Copyright ©The Author(s) 2000. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Dec 15, 2000; 6(6): 833-841
Published online Dec 15, 2000. doi: 10.3748/wjg.v6.i6.833
A high frequency of GBV-C/HGV coinfection in hepatitis C patients in Germany
Jie Yan, Reinhard H. Dennin
Jie Yan, Department of Pathogenic Biology, Medical School of Zhejiang University, Hangzhou 310006, Zhejiang Province, China
Reinhard H. Dennin, Institute of Medical Microbiology, Medical University of Leubeck, German y
Jie Yan, graduated from Zhejiang Medical University with Bachelor degree of medicine in 1982, obtained master degree of medicine from Zhejiang Medical University in 1987, and engaged in medical microbiological research, now as professor of microbiology, having 53 papers and 5 monographs published.
Author contributions: All authors contributed equally to the work.
Correspondence to: Jie Yan, Department of Pathogenic Biology, Medical School of Zhejiang University, Hangzhou 310006, Zhejiang Province, China. Email: yanchen@mail.hz.zj.cn
Telephone: 0086-571-7217385, Fax. 0086-571-7217044
Received: April 24, 2000
Revised: May 5, 2000
Accepted: May 12, 2000
Published online: December 15, 2000
Abstract

AIM: To detect infection rate of GBV-C/HGV in hepatitis C patients, to determine the methods of higher sensitivity and the primers of higher efficiency for GBV-C/HGV RNA detection and to study the dominant subtype and mutation of GBV-C/HGV.

METHODS: Quantitative RT-PCR for detection pf HCV RNA concentration in serum samples, RT-nested PCR with two sets of primers for detection of GBV-CRNA, RT-PCR ELISA with two sets of primers for detection of HGV RNA, nucleotide sequence and putative amino acid sequence analysis.

RESULTS: The positive rates of GBV-C RNA at the 5’-NCR and NS3 region in 211 serums amples from the patients with HCV infection were 31.8% and 22.8% respectively. The positive rates of HGV RNA at the 5’-NCR and NS5 region in the same samples were 47.9% and 31.8% respectively. The total positive rate of GBV-C/HGV RNA was as high as 55.5%. HCV copy numbers in the patients without GBV-C/HGV coinfection were statistically higher than that in the patients with GBV-C/HGV coinfection (P < 0.01). Frequent mutation of nucleotide residue was present in the amplification products. Frameshift mutation was found in two samples with GBV-C NS3 region nucleotide sequences. All nucleotide sequences from amplification products showed higher homology to HGV genome than to GBV-C genome even though part of the sequences were amplified with GBV-C primers.

CONCLUSION: A high frequency of GBV-C/HGV coinfection existed in the hepatitis C patients. RT-PCR ELISA was more sensitive than RT-nested PCR for detection of GBV-C/HGV RNA. The primers derived from the 5’-NCR was more efficient than those derived from the NS3 and NS5 regions. A reverse relationship was found to exist between HCV RNA concentration and GBV-C/HGV infection frequency. HGV was the dominant subtype of the virus in the local area. The major mutations of GBV-C/HGV genomes were random mutation of nucleotide residue.

Keywords: GB Virus C; hepatitis G virus; hepatitis C virus; coinfection; polymerase chain reaction; sequencing; dominant viral subtype, Germany