Original Articles
Copyright ©The Author(s) 1998. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Dec 15, 1998; 4(6): 506-510
Published online Dec 15, 1998. doi: 10.3748/wjg.v4.i6.506
Sequencing of p53 mutation in established human hepatocellular carcinoma cell line of HHC4 and HHC15 in nude mice
Shan-Min Yang, Hong Zhou, Rui-Chuan Chen, Yu-Fang Wang, Fu Chen, Chang-Gong Zhang, Yun Zhen, Jiang-Hua Yan, Jin-Hua Su
Shan-Min Yang, Hong Zhou, Rui-Chuan Chen, Yu-Fang Wang, Fu Chen, Chang-Gong Zhang, Yun Zhen, Jiang-Hua Yan, Jin-Hua Su, Cancer Research Center, Xiamen University, Xiamen 361005, Fujian, China
Shan-Min Yang, male, born on 1949-12-17 in Xiamen City, Han nationality, graduated from Fujian Medical University, director of Department of Cell Biology, associate professor of Cell Biology, major in Cell Biology of Tumor, having 50 papers published.
Author contributions: All authors contributed equally to the work.
Correspondence to: Dr. Shan-Min Yang, Cancer Research Center, Xiamen University, Xiamen 361005, Fujian, China
Telephone: +86-592-2017309
Received: November 9, 1998
Revised: November 22, 1998
Accepted: December 5, 1998
Published online: December 15, 1998
Abstract

AIM: To set up cell lines of human hepatocellular carcinoma in nude mice for the research of cell biology and gene therapy.

METHODS: Xenotransplantation of human hepatoma into nude mice was carried out and the growth rate, histopathology and immunology of the nude mice were studied. The DNA from xenografts were analyzed by HBV gene and PCR amplification of a fragment of p53 gene exon 7, which were identified by dot blot hybridization, restriction fragments length polymorphism and DNA sequencing.

RESULTS: HHC4 and hHCC415 cell lines could be successively transplanted in nude mice and the population doubling time was 7 and 5 d respectively. These strains retained the original characteristics of histopathology, secreting AFP and heteroploid karyotypes in human hepatocellular carcinoma. The fragment of HBV gene was detected in the genomic DNA of both hHCC4 and hHCC15, however only hHCC4 secreted HBsAg. The mutation at 250 code (C→A) and 249 code (G→T) were detected respectively in the genomic DNA of HHC4 and HHC15.

CONCLUSION: The two cell lines are useful material for the studying of cell biology and gene therapy in human hepatocellular carcinoma and provide molecular biological trace of the relationship between high mortality of hepatoma and AFB1 severe pollution of the daily common foods in this district.

Keywords: Liver neoplasms; carcinoma, hepatocellular; p53 gene; mutation; HHC4; HHC15; Tumor cell, cultured