Published online Jan 7, 2025. doi: 10.3748/wjg.v31.i1.99954
Revised: October 24, 2024
Accepted: November 19, 2024
Published online: January 7, 2025
Processing time: 127 Days and 16.8 Hours
Transfer RNA (tRNA)-derived fragments, a new type of tRNA-derived small RNA (tsRNA), can be cleaved from tRNA by enzymes to regulate target gene expression at the transcriptional and translational levels. tsRNAs are not only degradation fragments but also have biological functions, including those in immune inflammation, metabolic disorders, and cell death. tsRNA dysregulation is closely associated with multiple diseases, including various cancers and acute pancreatitis (AP). AP is a common gastrointestinal disease, and its incidence increases annually. AP development is associated with tsRNAs, which regulate cell injury and induce inflammation, especially pyroptosis and ferroptosis. Notably, serum tRF36 has the potential to serve as a non-invasive diagnostic biomarker and leads to pancreatic acinar cell ferroptosis causing inflammation to promote AP. We show the characteristics of tsRNAs and their diagnostic value and function in AP, and discuss the potential opportunities and challenges of using tsRNAs in clinical applications and research.
Core Tip: The dysregulated expression of tRNA-derived small RNAs (tsRNAs) in acute pancreatitis (AP) reveals the molecular mechanism by which tsRNAs affect the occurrence and development of AP, and can be used as diagnostic markers and potential therapeutic targets. In cell and animal models, the inhibition or overexpression of tsRNA has been shown to influence AP development. The results showed that tsRNAs accelerated AP progression by inducing an increase in inflammatory factors and promoting cell death (including pyroptosis and ferroptosis).