Published online Oct 7, 2017. doi: 10.3748/wjg.v23.i37.6907
Peer-review started: February 27, 2017
First decision: April 21, 2017
Revised: May 12, 2017
Accepted: July 12, 2017
Article in press: July 12, 2017
Published online: October 7, 2017
Processing time: 215 Days and 12 Hours
Magnesium deficiency can cause a variety of symptoms, including potentially life-threatening complications such as seizures, cardiac arrhythmias and secondary electrolyte disturbances. Hypomagnesemia can be a serious adverse effect to proton pump inhibitor (PPI) therapy, which is worrying due to the widespread use of PPIs. Current evidence suggest that the mechanism of PPI induced hypomagnesemia is impaired intestinal magnesium absorption. In this report, we present the case of a long-term PPI user with persistent hypomagnesemia with severe symptoms at presentation. He was unable to stop PPI treatment because of severe reflux symptoms, and was dependent on weekly intravenous magnesium infusions, until his magnesium levels finally normalized without the need for supplementation after a successful laparoscopic fundoplication.
Core tip: This case illustrates a long-term proton pump inhibitor (PPI) user with persistent hypomagnesaemia causing severe symptoms at presentation. For years, he was dependent on oral and later intravenous magnesium supplementation, until his hypomagnesemia rapidly improved upon PPI discontinuation. He was, however, not able to cope with the resulting reflux symptoms, and was not able to completely stop PPI treatment until he underwent a successful laparoscopic fundoplication.