Published online Jul 28, 2015. doi: 10.3748/wjg.v21.i28.8508
Peer-review started: January 22, 2015
First decision: March 26, 2015
Revised: April 24, 2015
Accepted: May 27, 2015
Article in press: May 27, 2015
Published online: July 28, 2015
Processing time: 189 Days and 18.2 Hours
Although the outcome of newborns with surgical congenital diseases (e.g., diaphragmatic hernia; esophageal atresia; omphalocele; gastroschisis) has improved rapidly with recent advances in perinatal intensive care and surgery, infant survivors often require intensive treatment after birth, have prolonged hospitalizations, and, after discharge, may have long-term sequelae including gastro-intestinal comorbidities, above all, gastroesophageal reflux (GER). This condition involves the involuntary retrograde passage of gastric contents into the esophagus, with or without regurgitation or vomiting. It is a well-recognized condition, typical of infants, with an incidence of 85%, which usually resolves after physiological maturation of the lower esophageal sphincter and lengthening of the intra-abdominal esophagus, in the first few months after birth. Although the exact cause of abnormal esophageal function in congenital defects is not clearly understood, it has been hypothesized that common (increased intra-abdominal pressure after closure of the abdominal defect) and/or specific (e.g., motility disturbance of the upper gastrointestinal tract, damage of esophageal peristaltic pump) pathological mechanisms may play a role in the etiology of GER in patients with birth defects. Improvement of knowledge could positively impact the long-term prognosis of patients with surgical congenital diseases. The present manuscript provides a literature review focused on pathological and clinical characteristics of GER in patients who have undergone surgical treatment for congenital abdominal malformations.
Core tip: Although the outcome of newborns with surgical congenital diseases has improved rapidly with recent advances in perinatal intensive care and surgery, infant survivors often may have long-term sequelae including, above all, gastroesophageal reflux (GER). Common or specific pathological mechanisms may play a role in the etiology of GER in patients with birth defects. The improvement of knowledge of long-term outcome and follow-up could positively impact the long-term prognosis of newborns with surgical congenital diseases. The present manuscript provides a literature review focused on pathological and clinical characteristics of GER in patients who have undergone surgical treatment for congenital abdominal malformations.