Published online Jun 28, 2014. doi: 10.3748/wjg.v20.i24.7849
Revised: February 15, 2014
Accepted: March 19, 2014
Published online: June 28, 2014
Processing time: 210 Days and 12.7 Hours
The treatment of advanced pancreatic cancer has not moved much beyond single agent gemcitabine until recently when protocols such as FOLFIRINOX (fluorouracil, leucovorin, irinotecan and oxaliplatin) and nab-paclitaxel-gemcitabine have demonstrated some improved outcomes. Advances in technology especially in massively parallel genome sequencing has progressed our understanding of the biology of pancreatic cancer especially the candidate signalling pathways that are involved in tumourogenesis and disease course. This has allowed identification of potentially actionable mutations that may be targeted by new biological agents. The heterogeneity of pancreatic cancer makes tumour tissue collection important with the aim of being able to personalise therapies for the individual as opposed to a one size fits all approach to treatment of the condition. This paper reviews the developments in this area of translational research and the ongoing clinical studies that will attempt to move this into the everyday oncology practice.
Core tip: State of art review of genomic developments in pancreatic cancer that will hopefully lead to a new treatment paradigm of recognising that pancreatic cancer is a heterogenous disease. Adequate tissue collection is important to allow biomarker testing and molecular sequencing to allow determination of actionable mutations so that personalised therapies can be used in a rational manner.