Case Report
Copyright ©2012 Baishideng Publishing Group Co., Limited. All rights reserved.
World J Gastroenterol. Mar 14, 2012; 18(10): 1137-1140
Published online Mar 14, 2012. doi: 10.3748/wjg.v18.i10.1137
Large-vessel thrombosis in intestinal Behçet’s disease complicated with myelodysplastic syndrome and trisomy 8
Huang-Chi Chen, Ying-Ming Chiu
Huang-Chi Chen, Division of Critical Care Medicine, Department of Internal Medicine, Changhua Christian Hospital, 500 Changhua, Taiwan, China
Ying-Ming Chiu, Division of Allergy, Immunology and Rheumatology, Department of Internal Medicine, Changhua Christian Hospital, 500 Changhua, Taiwan, China
Author contributions: Chen HC and Chiu YM contributed equally to this work; Chen HC collected the data and drafted the manuscript; Chiu YM critically revised the manuscript’s intellectual content.
Correspondence to: Ying-Ming Chiu, MD, Division of Allergy, Immunology and Rheumatology, Department of Internal Medicine, Changhua Christian Hospital, 135 Nanxiao Street, Changhua City, 500 Changhua, Taiwan, China. 129273@cch.org.tw
Telephone: +886-4-7238595 Fax: +886-4-7228289
Received: February 21, 2011
Revised: June 24, 2011
Accepted: January 18, 2012
Published online: March 14, 2012
Abstract

Behçet’s disease is characterized by recurrent oral ulcers, genital ulcers, uveitis and skin lesions. Myelodysplastic syndrome (MDS) is characterized by problems due to ineffective hematopoiesis. Several studies have identified a relationship between MDS and Behçet’s disease, especially intestinal Behçet’s disease. Trisomy 8 seems to play an important role in these disorders as well. The present case was a 24-year-old woman who had a huge tonsil ulcer with initial symptoms of odynophagia and intermittent fever. We also noted folliculitis on her upper back. Five days later, she began to experience diarrhea and abdominal pain. Abdominal computed tomography and subsequent surgery revealed ileum perforation and enterocolitis with multiple ulcers. Later, she was admitted again for a vulvar suppurative ulcer and suspicious Bartholin’s cyst infection. The patient’s clinical presentations met the criteria for Behçet’s disease. Six months after the bowel perforation event, we noted the development of pancytopenia in a routine laboratory examination. All the examinations led to the diagnosis of MDS with trisomy 8. The most unusual finding was that multiple large vessel thrombi developed during follow-up. Previous studies have suggested that trisomy 8 in MDS leads to concurrent intestinal Behçet’s disease. Moreover, the inflammatory and immune genes related to thrombus formation are overexpressed in cases of MDS with trisomy 8. Trisomy 8 must play a role in thrombosis. Further studies are needed to help clarify the pathophysiology and pathogenesis of these disorders.

Keywords: Behçet’s disease; Myelodysplastic syndrome; Trisomy 8; Intestinal ulcers; Thrombosis