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©2007 Baishideng Publishing Group Inc.
World J Gastroenterol. Dec 7, 2007; 13(45): 5989-5994
Published online Dec 7, 2007. doi: 10.3748/wjg.v13.i45.5989
Published online Dec 7, 2007. doi: 10.3748/wjg.v13.i45.5989
Figure 2 Histological assessment of livers following death or living for 15 d (original magnification, × 100, A, B; × 400, C, D, E).
Hepatocellular bridged necrosis, mononuclear and lymphoid inflammatory infiltration, necrosis and hemorrhage (A) accompanied reticular fibers structures collapsing in control group (B). Increased mononuclear and lymphoid inflammatory infiltration, and necrosis in groups OAT, O3, and YC (C, D) compared with group O3 + YC (E). Inflammation and necrosis were graded on a 4-point scale (Table 1) by a blinded pathologist in 20 random high power fields per animal (n = 6 in each group), represented graphically in panels (F) errorbars: 95.00% CI and (G) errorbars: 95.00% CI. Data represent mean ± SE; aP < 0.05 vs Control.
- Citation: Li LJ, Yang YG, Zhang ZL, Nie SF, Li Z, Li F, Hua HY, Hu YJ, Zhang HS, Guo YB. Protective effects of medical ozone combined with traditional Chinese medicine against chemically-induced hepatic injury in dogs. World J Gastroenterol 2007; 13(45): 5989-5994
- URL: https://www.wjgnet.com/1007-9327/full/v13/i45/5989.htm
- DOI: https://dx.doi.org/10.3748/wjg.v13.i45.5989