Observational Study
Copyright ©The Author(s) 2022.
World J Clin Cases. Jan 7, 2022; 10(1): 217-226
Published online Jan 7, 2022. doi: 10.12998/wjcc.v10.i1.217
Figure 1
Figure 1 Coronal portal venous phase computed tomography demonstrating a cirrhotic liver with ascites, varices, and superior mesenteric vein thrombosis. Anticoagulation was discontinued due to upper gastrointestinal hemorrhage. As such, the patient underwent endovascular interventions. A: Before the thrombectomy and transjugular intrahepatic portosystemic shunt clinical target volume interventions; B: Post-intervention.
Figure 2
Figure 2 Laboratory tests, imaging, and treatment modalities. A and B: Selective superior mesenteric venogram after transjugular intrahepatic portosystemic shunt creation demonstrates a filling defect representing thrombus in the superior mesenteric venous (SMV), hepatofugal flow in a peripheral SMV branch, and a venovenous collateral decompressing the small bowel outflow into the main SMV trunk; C: Superior mesenteric venogram after aspiration and mechanical thrombectomy demonstrates reestablished flow in the previously occluded SMV, nonperfusion for the venovenous collateral, and hepatopetal flow in the peripheral SMV branches.
Figure 3
Figure 3 Kaplan-Meier curve of primary patency.