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World J Nephrol. Feb 6, 2015; 4(1): 98-104
Published online Feb 6, 2015. doi: 10.5527/wjn.v4.i1.98
Role of β2-microglobulin in uremic patients may be greater than originally suspected
Aysegul Zumrutdal
Aysegul Zumrutdal, Nephrology Department, Baskent University Adana Teaching and Research Center, Baskent University Hospital, Yuregir, Adana 01230, Turkey
Author contributions: Zumrutdal A solely contributed to this work.
Open-Access: This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/
Correspondence to: Aysegul Zumrutdal, MD, Professor, Nephrology Department, Baskent University Adana Teaching and Research Center, Baskent University Hospital, Yuregir, Dadaloglu Mah, 2591 St, 4/A, Adana 01230, Turkey. azumrutdal@yahoo.com
Telephone: +90-322-3272727 Fax: +90-322-3271274
Received: June 17, 2014
Peer-review started: June 18, 2014
First decision: August 14, 2014
Revised: September 3, 2014
Accepted: October 1, 2014
Article in press: October 10, 2014
Published online: February 6, 2015
Processing time: 234 Days and 17.8 Hours
Core Tip

Core tip: Previously, the clinical significance of beta2-microglobulin (β2M) in uremic patients was limited to β2M-derived amyloidosis; in recent years, its role and power has changed and expanded. Although there have been some inconsistencies among the various analyses relating to β2M, the data generally support β2M as a promising novel marker of kidney function by predicting cardiovascular (CV) risk, CV events and overall mortality.