Peer-review started: June 7, 2015
First decision: August 16, 2015
Revised: September 5, 2015
Accepted: November 13, 2015
Article in press: November 17, 2015
Published online: January 6, 2016
Processing time: 217 Days and 10 Hours
The clinical spectrum of human immunodeficiency virus (HIV) infection associated disease has changed significantly over the past decade, mainly due to the wide availability and improvement of combination antiretroviral therapy regiments. Serious complications associated with profound immunodeficiency are nowadays fortunately rare in patients with adequate access to care and treatment. However, HIV infected patients, and particularly those with acquired immune deficiency syndrome, are predisposed to a host of different water, electrolyte, and acid-base disorders (sometimes with opposite characteristics), since they have a modified renal physiology (reduced free water clearance, and relatively increased fractional excretion of calcium and magnesium) and they are also exposed to infectious, inflammatory, endocrinological, oncological variables which promote clinical conditions (such as fever, tachypnea, vomiting, diarrhea, polyuria, and delirium), and may require a variety of medical interventions (antiviral medication, antibiotics, antineoplastic agents), whose combination predispose them to undermine their homeostatic capability. As many of these disturbances may remain clinically silent until reaching an advanced condition, high awareness is advisable, particularly in patients with late diagnosis, concomitant inflammatory conditions and opportunistic diseases. These disorders contribute to both morbidity and mortality in HIV infected patients.
Core tip: Human immunodeficiency virus infected patients, and particularly those with acquired immune deficiency syndrome, are predisposed to different water, electrolyte, and acid-base disorders since they have a modified renal physiology and they also are exposed to infectious, inflammatory, endocrinological, oncological, and pharmacological variables whose combination undermine their homeostatic capability. We herein discuss each of these internal milieu alterations usually observed in this group.