Published online Aug 19, 2021. doi: 10.5498/wjp.v11.i8.463
Peer-review started: February 24, 2021
First decision: July 4, 2021
Revised: July 12, 2021
Accepted: July 29, 2021
Article in press: July 29, 2021
Published online: August 19, 2021
Processing time: 169 Days and 3.8 Hours
Schizophrenia is a chronic psychiatric condition consisting of positive and negative symptoms causing significant impacts on life. Current treatment includes second-generation antipsychotics (SGAs), the use of which is associated with side effects including: increased metabolic risk, sleep dysfunction and extrapyramidal side effects (EPS). Exogenous melatonin has been demonstrated to attenuate sleep dysfunction in the general population, however its indication in schizophrenia has been relatively unexplored. The proven antioxidant and neuroprotective effects of melatonin suggest potential for therapeutic benefit in adjunctive treatment of schizophrenia, especially in attenuating side effects associated with SGAs.
Current therapeutic treatment for schizophrenia often causes side effects including increased cardiovascular and metabolic risk, sleep dysfunction and EPS. Adjunctive use of melatonin has been suggested to benefit sleep disturbances, however its indication in schizophrenia has remained unclear. Therefore, we synthesized the current evidence for the effect of adjunctive use of melatonin on any outcome in individuals with schizophrenia.
To synthesize clinical trials conducted to date that have investigated the use of melatonin as an adjunctive therapy for individuals with schizophrenia in improving any therapeutic outcome.
A systematic literature search was conducted on MEDLINE (Ovid), Embase, PsychINFO, PubMed, CINAHL and Cochrane Library for clinical trials using melatonin as an adjunctive therapy that included a group of patients with schizophrenia. PRISMA guidelines were adhered to and the Cochrane risk-of-bias tool for randomized controlled trials was used by two authors to assess the trials independently, with consensus confirmed by a third reviewer.
A total of 15 trials were included for qualitative synthesis after assessing for eligibility and removing duplicates. The trials assessed the following primary outcomes: sleep (n = 6), metabolic profile (n = 3), tardive dyskinesia (n = 3), cognitive function (n = 2) and benzodiazepine discontinuation (n = 1).
Positive outcomes were demonstrated for the use of melatonin in improving sleep efficiency and certain metabolic outcomes, specifically in first-episode patients initiating antipsychotic treatment. Currently, there is limited therapeutic indication for the use of melatonin in treatment of tardive dyskinesia, cognitive function or facilitating benzodiazepine discontinuation. Limitations included small sample sizes and no standardization of the duration and/or dosage of adjunctive melatonin used.
Future studies are required to confirm these improvements, determine the pharmacokinetic interactions of melatonin with specific antipsychotic medications and develop a standardized duration and dosage of adjunctive melatonin treatment. Moreover, a long-term safety and efficacy profile remains to be determined.