Published online Aug 4, 2015. doi: 10.5492/wjccm.v4.i3.163
Peer-review started: February 9, 2015
First decision: April 10, 2015
Revised: May 8, 2015
Accepted: May 27, 2015
Article in press: May 28, 2015
Published online: August 4, 2015
Processing time: 190 Days and 0 Hours
Major pulmonary disorders may occur after brain injuries as ventilator-associated pneumonia, acute respiratory distress syndrome or neurogenic pulmonary edema. They are key points for the management of brain-injured patients because respiratory failure and mechanical ventilation seem to be a risk factor for increased mortality, poor neurological outcome and longer intensive care unit or hospital length of stay. Brain and lung strongly interact via complex pathways from the brain to the lung but also from the lung to the brain. Several hypotheses have been proposed with a particular interest for the recently described “double hit” model. Ventilator setting in brain-injured patients with lung injuries has been poorly studied and intensivists are often fearful to use some parts of protective ventilation in patients with brain injury. This review aims to describe the epidemiology and pathophysiology of lung injuries in brain-injured patients, but also the impact of different modalities of mechanical ventilation on the brain in the context of acute brain injury.
Core tip: Brain lung crosstalk is a complex interaction from the brain to the lung but also from the lung to the brain. Intensivists are often fearful to use some parts of protective ventilation in patients with brain injuries but if correctly applied, mechanical ventilation could have beneficial effect on brain oxygenation, even if positive end-expiratory pressure and recruitment maneuvers are used. This review aims to describe the epidemiology and pathophysiology of lung injuries in brain-injured patients, but also the impact of different modalities of mechanical ventilation on the brain in the context of acute brain injury.