Published online Jul 27, 2015. doi: 10.5313/wja.v4.i2.44
Peer-review started: January 21, 2015
First decision: February 7, 2015
Revised: March 31, 2015
Accepted: April 27, 2015
Article in press: April 29, 2015
Published online: July 27, 2015
Processing time: 188 Days and 5.4 Hours
We present the case of a 13-year-old boy undergoing scoliosis repair utilizing skull-femoral traction who developed sudden, sustained bradycardia and hypotension during scoliosis repair, associated with loss of somatosensory evoked potentials and motor evoked potentials to all four limbs. A diagnosis of spinal shock and hypovolemia was made after ruling out primary cardiac causes, sepsis, anaphylaxis and intra-spinal pedicle screw placement. Acute complications of surgical scoliosis repair are reviewed along with anatomy of the sympathetic nervous system. In this case spinal shock may have been due to hypovolemia as well as spinal cord manipulation during T12 vertebral column resection that was needed to effect scoliosis correction. Treatment included volume expansion and inotropic support. Anesthesiologists caring for these patients should be mindful of the possibility of spinal shock during correction of severe scoliosis, particularly when vertebral column resection is undertaken.
Core tip: A child undergoing scoliosis repair developed sudden bradycardia and hypotension, associated with loss of somatosensory and motor evoked potentials to all four limbs. Spinal shock and hypovolemia were diagnosed after ruling out other causes. Acute complications of scoliosis repair are reviewed along with sympathetic nervous system anatomy. Spinal shock was likely due to hypovolemia and spinal cord manipulation during vertebral column resection that was needed to effect scoliosis correction. Treatment included volume expansion and inotropic support. Anesthesiologists should be mindful of the possibility of spinal shock during correction of severe scoliosis, particularly when vertebral column resection is undertaken.